YEA . . . What he said !!Lucky_P":3iecf4fp said:As has been typical, your reasoning is flawed, and without logic.
In monogastrics, sulfur is essentially inert. Baby calves are monogastrics, with no rumen function.
Even IF the dam were consuming enough excessive S - or excessive grain, both of which can precipitate polioencephalomalacia - and you've NOT demonstrated that the dam of this calf was affected - any S that MIGHT be passing to this calf will have no detrimental effect.
The 'polio-like' deal in CA children is a viral poliomyelitis, and has no connection whatsoever to polioenceophalomalacia in cattle, much less any association with what may or may not have been fed to the cattle that may or may not have produced beef that these affected children may or may not have consumed.
The associations that you're attempting to draw are no more plausible than saying that because cattle sometimes die of blackleg, that teenagers develop blackheads(comedones, plugged pores, zits) as a result of eating beef - and are at risk of dying.
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