talltimber":bw5pyu9p said:
Mine usually start to scatter somewhere around the beginning of the swoop stage.
There is a fella in my area with two cases of it a month or so ago. Lost them both. One, he felt like he caught it early enough. She lasted a few days. He was putting ctc in mineral, backrubbers, spraying occasionally.
Darin Stanfield, DVD, stated that no packaged mineral that he reviewed had even close to the level of chlorotetracycline (ctc) to protect against anaplasmosis. He attempted to work out an arrangement with the Hinton Mills company in this region for a custom mineral that provided enough CTC to meet the recommended level. Hinton Mills said it was cost prohibitive. I don't have the number handy but the level of CTC needed in mineral to be effective was several orders of magnitude above the level in most packaged mineral.
PS: minor point. Notice in the reference HD provided, it is an infectious disease of the RBCs. Caused by a reckesttsial
parasite.
At least when I was in graduate school, reckesttsials were not treated as bacteria in the same fashion as say Clostridials. Clostridials cause Blackleg and tetanus, etc. You do not refer to Clostridium as a parasite. You do refer to trichomonas as a parasite of the Bull's sheath. Parasites require a live host. Reckesttials function as a parasite.
Edited to add: there is a PDF file on anaplasmosis in beef cattle put out by Texas A&M. I cannot get the URL to come up. But it lists horse flies, stable flies and mosquitos as significant vectors. Says recent studies are indicating ticks are more significant than once believed.