Boondock, that linked article is nearly 25 years old... it reflected our knowledge (or what we thought we knew) at that time... some of the information is valid, but there's a lot in there that is now known not to be correct.
Clinical disease is seen mostly in mature animals.
We rarely see clinical cases of anaplasmosis in cattle under 12-15-18 months of age - they're still building body mass, their erythropoietic (red blood cell-making) system is still ramped up - so they respond immunologically and hematopoietically, and can rapidly overcome the loss of RBCs without becoming severely anemic. In some of those older calves, they may have increased temperature and increased respiratory rate due to borderline anemia - and be misdiagnosed as respiratory disease cases.
These animals which are infected as youngsters will be persistently infected, and can serve as a source of infection for other animals in the herd... but will likely never develop clinical disease.
'Naive' older animals that were not exposed early in life are the ones that are most at risk; if introduced into a herd (or area) where infected animals are present - or if an infected animal is introduced into a herd with no previous infection...trouble may not be far off.
Feeding CTC at 0.5mg/lb/day during the vector(ticks) season will control MOST clinical cases in infected herds - will NOT prevent infection, and there may be an occasional 'breakthrough' case.
Medicated minerals may help, but even if each animal consumes the theoretical 4 oz of mineral per day that most are formulated for... there's only enough CTC in there for a 750 lb animal... May not be enough to keep from having clinical cases... but better than nothing.
Back in the past, we'd often (mistakenly) recommend treating the entire herd with an LA-oxytet... but sometimes we just prolonged the outbreak; the infection in those animals in the incubation phase just picked right back up where it left off when the antibiotics wore off. Saw one herd one time that prolonged the outbreak from August, clear into late December by wholesale treating with LA OTC...
Current recommendations are to treat clinically-affected animals with injectible OTC and start feeding CTC to the herd.
Treating clinically-affected animals with injectible OTC just slows the organism down long enough (hopefully) for the animal to ramp up RBC production enough to survive the acute phase.
Treatment with oxytet (even multiple doses) will not effectively 'clear' the infection... animals that survive will be persistently infected, and can continue to serve as a source of infection.
Feeding CTC at 2mg/lb/day for 60 days will 'clear' most animals of the infection - but they are susceptible to reinfection. I don't know that I could recommend 'feeding for clearance' for most operations... but if they HAVE to have test-negative animals for sale, it may be necessary.
While anaplasmosis is principally tick-vectored in most areas, it can be spread by needles, instruments, OB sleeves, etc., and theoretically, by biting flies - though other than with strains of the organism found mostly in FL and CA - horseflies, etc. are of minimal importance...
I occasionally see cases far outside the normal expected time frame - say, like in March - and have to think that those were probably iatrogenic(human-caused)... and that those producers 'worked the herd' 6 weeks or so earlier and transmitted it from a carrier animal to a naive one by reusing needles between multiple animals.
There is a vaccine available... currently licensed for sale in something like 18 states. Reports I've received from vets/producers using it have been favorable. Does not prevent infection, and vaccinated animals will be seropositive... but it seems to all but eliminate clinical cases.