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TEXAS CONFIRMED HUMAN BSE VCJD IN ADULT MALE
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<blockquote data-quote="flounder" data-source="post: 1165826" data-attributes="member: 3519"><p>CJD </p><p></p><p><a href="http://www.plosone.org/annotation/listThread.action?root=363" target="_blank">http://www.plosone.org/annotation/listT ... n?root=363</a> </p><p></p><p>Views & Reviews</p><p></p><p>Monitoring the occurrence of emerging forms of Creutzfeldt-Jakob disease in the United States</p><p></p><p>Ermias D. Belay, MD, Ryan A. Maddox, MPH, Pierluigi Gambetti, MD and Lawrence B. Schonberger, MD</p><p></p><p>+ Author Affiliations</p><p></p><p>From the Division of Viral and Rickettsial Diseases (Drs. Belay and Schonberger and R.A. Maddox), National Center for Infectious Diseases, Centers for Disease Control and Prevention, Atlanta, GA; and National Prion Disease Pathology Surveillance Center (Dr. Gambetti), Division of Neuropathology, Institute of Pathology, Case Western Reserve University, Cleveland, OH.</p><p></p><p>Address correspondence and reprint requests to Dr. Ermias D. Belay, 1600 Clifton Road, Mailstop A-39, Atlanta, GA 30333. </p><p></p><p><a href="http://www.neurology.org/content/60/2/176" target="_blank">http://www.neurology.org/content/60/2/176</a> </p><p></p><p>26 March 2003</p><p></p><p>Terry S. Singeltary, retired (medically) CJD WATCH</p><p></p><p>I lost my mother to hvCJD (Heidenhain Variant CJD). I would like to comment on the CDC's attempts to monitor the occurrence of emerging forms of CJD. Asante, Collinge et al [1] have reported that BSE transmission to the 129-methionine genotype can lead to an alternate phenotype that is indistinguishable from type 2 PrPSc, the commonest sporadic CJD. However, CJD and all human TSEs are not reportable nationally. CJD and all human TSEs must be made reportable in every state and internationally. I hope that the CDC does not continue to expect us to still believe that the 85%+ of all CJD cases which are sporadic are all spontaneous, without route/source. We have many TSEs in the USA in both animal and man. CWD in deer/elk is spreading rapidly and CWD does transmit to mink, ferret, cattle, and squirrel monkey by intracerebral inoculation. With the known incubation periods in other TSEs, oral transmission studies of CWD may take much longer. Every victim/family of CJD/TSEs should be asked about route and source of this agent. To prolong this will only spread the agent and needlessly expose others. In light of the findings of Asante and Collinge et al, there should be drastic measures to safeguard the medical and surgical arena from sporadic CJDs and all human TSEs. I only ponder how many sporadic CJDs in the USA are type 2 PrPSc? </p><p></p><p><a href="http://www.neurology.org/content/60/2/176/reply#neurology_el_535" target="_blank">http://www.neurology.org/content/60/2/1 ... ogy_el_535</a></p><p></p><p>Diagnosis and Reporting of Creutzfeldt-Jakob Disease </p><p></p><p>Singeltary, Sr et al. JAMA.2001; 285: 733-734. Vol. 285 No. 6, February 14, 2001 JAMA</p><p></p><p>Diagnosis and Reporting of Creutzfeldt-Jakob Disease</p><p></p><p>To the Editor: In their Research Letter, Dr Gibbons and colleagues1 reported that the annual US death rate due to Creutzfeldt-Jakob disease (CJD) has been stable since 1985. These estimates, however, are based only on reported cases, and do not include misdiagnosed or preclinical cases. It seems to me that misdiagnosis alone would drastically change these figures. An unknown number of persons with a diagnosis of Alzheimer disease in fact may have CJD, although only a small number of these patients receive the postmortem examination necessary to make this diagnosis. Furthermore, only a few states have made CJD reportable. Human and animal transmissible spongiform encephalopathies should be reportable nationwide and internationally.</p><p></p><p>Terry S. Singeltary, Sr Bacliff, Tex</p><p></p><p>1. Gibbons RV, Holman RC, Belay ED, Schonberger LB. Creutzfeldt-Jakob disease in the United States: 1979-1998. JAMA. 2000;284:2322-2323. FREE FULL TEXT </p><p></p><p><a href="http://jama.ama-assn.org/cgi/content/full/285/6/733?maxtoshow=&HITS=10&hits=10&RESULTFORMAT=&fulltext=singeltary&searchid=1&FIRSTINDEX=0&resourcetype=HWCIT" target="_blank">http://jama.ama-assn.org/cgi/content/fu ... type=HWCIT</a> </p><p></p><p>2 January 2000</p><p></p><p>British Medical Journal</p><p></p><p>U.S. Scientist should be concerned with a CJD epidemic in the U.S., as well</p><p></p><p><a href="http://www.bmj.com/cgi/eletters/320/7226/8/b#6117" target="_blank">http://www.bmj.com/cgi/eletters/320/7226/8/b#6117</a> </p><p></p><p>15 November 1999</p><p></p><p>British Medical Journal</p><p></p><p>vCJD in the USA * BSE in U.S.</p><p></p><p><a href="http://www.bmj.com/cgi/eletters/319/7220/1312/b#5406" target="_blank">http://www.bmj.com/cgi/eletters/319/7220/1312/b#5406</a></p></blockquote><p></p>
[QUOTE="flounder, post: 1165826, member: 3519"] CJD [url=http://www.plosone.org/annotation/listThread.action?root=363]http://www.plosone.org/annotation/listT ... n?root=363[/url] Views & Reviews Monitoring the occurrence of emerging forms of Creutzfeldt-Jakob disease in the United States Ermias D. Belay, MD, Ryan A. Maddox, MPH, Pierluigi Gambetti, MD and Lawrence B. Schonberger, MD + Author Affiliations From the Division of Viral and Rickettsial Diseases (Drs. Belay and Schonberger and R.A. Maddox), National Center for Infectious Diseases, Centers for Disease Control and Prevention, Atlanta, GA; and National Prion Disease Pathology Surveillance Center (Dr. Gambetti), Division of Neuropathology, Institute of Pathology, Case Western Reserve University, Cleveland, OH. Address correspondence and reprint requests to Dr. Ermias D. Belay, 1600 Clifton Road, Mailstop A-39, Atlanta, GA 30333. [url=http://www.neurology.org/content/60/2/176]http://www.neurology.org/content/60/2/176[/url] 26 March 2003 Terry S. Singeltary, retired (medically) CJD WATCH I lost my mother to hvCJD (Heidenhain Variant CJD). I would like to comment on the CDC's attempts to monitor the occurrence of emerging forms of CJD. Asante, Collinge et al [1] have reported that BSE transmission to the 129-methionine genotype can lead to an alternate phenotype that is indistinguishable from type 2 PrPSc, the commonest sporadic CJD. However, CJD and all human TSEs are not reportable nationally. CJD and all human TSEs must be made reportable in every state and internationally. I hope that the CDC does not continue to expect us to still believe that the 85%+ of all CJD cases which are sporadic are all spontaneous, without route/source. We have many TSEs in the USA in both animal and man. CWD in deer/elk is spreading rapidly and CWD does transmit to mink, ferret, cattle, and squirrel monkey by intracerebral inoculation. With the known incubation periods in other TSEs, oral transmission studies of CWD may take much longer. Every victim/family of CJD/TSEs should be asked about route and source of this agent. To prolong this will only spread the agent and needlessly expose others. In light of the findings of Asante and Collinge et al, there should be drastic measures to safeguard the medical and surgical arena from sporadic CJDs and all human TSEs. I only ponder how many sporadic CJDs in the USA are type 2 PrPSc? [url=http://www.neurology.org/content/60/2/176/reply#neurology_el_535]http://www.neurology.org/content/60/2/1 ... ogy_el_535[/url] Diagnosis and Reporting of Creutzfeldt-Jakob Disease Singeltary, Sr et al. JAMA.2001; 285: 733-734. Vol. 285 No. 6, February 14, 2001 JAMA Diagnosis and Reporting of Creutzfeldt-Jakob Disease To the Editor: In their Research Letter, Dr Gibbons and colleagues1 reported that the annual US death rate due to Creutzfeldt-Jakob disease (CJD) has been stable since 1985. These estimates, however, are based only on reported cases, and do not include misdiagnosed or preclinical cases. It seems to me that misdiagnosis alone would drastically change these figures. An unknown number of persons with a diagnosis of Alzheimer disease in fact may have CJD, although only a small number of these patients receive the postmortem examination necessary to make this diagnosis. Furthermore, only a few states have made CJD reportable. Human and animal transmissible spongiform encephalopathies should be reportable nationwide and internationally. Terry S. Singeltary, Sr Bacliff, Tex 1. Gibbons RV, Holman RC, Belay ED, Schonberger LB. Creutzfeldt-Jakob disease in the United States: 1979-1998. JAMA. 2000;284:2322-2323. FREE FULL TEXT [url=http://jama.ama-assn.org/cgi/content/full/285/6/733?maxtoshow=&HITS=10&hits=10&RESULTFORMAT=&fulltext=singeltary&searchid=1&FIRSTINDEX=0&resourcetype=HWCIT]http://jama.ama-assn.org/cgi/content/fu ... type=HWCIT[/url] 2 January 2000 British Medical Journal U.S. Scientist should be concerned with a CJD epidemic in the U.S., as well [url=http://www.bmj.com/cgi/eletters/320/7226/8/b#6117]http://www.bmj.com/cgi/eletters/320/7226/8/b#6117[/url] 15 November 1999 British Medical Journal vCJD in the USA * BSE in U.S. [url=http://www.bmj.com/cgi/eletters/319/7220/1312/b#5406]http://www.bmj.com/cgi/eletters/319/7220/1312/b#5406[/url] [/QUOTE]
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