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Artificial Insemination (AI) for Cattle
In-breeding Risk same grandfather & great great grandfather
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<blockquote data-quote="Lucky_P" data-source="post: 1648573" data-attributes="member: 12607"><p>Nick, </p><p>Your supposition that this bull inherited the SCN defect isn't born out by scientific examination. </p><p>This bull we purchased was, as far as we can tell, the originator of the SCN defect; that is, the mutation arose in him. While his dam and sire were not available for testing when the defective gene sequence was characterized, AI sires several generations back were tested, and found to be free of the defect, as was the single half-sib cow remaining in the herd of origin. The bull was by a son of SAF Focus of ER, out of a New Design 878 daughter... well-known genetics to Angus folks, and most of the ancestors in the 4-5 generations behind him had material on file available for testing, and all were found to be free of the SCN defect. </p><p></p><p>For example, GAR Precision 1680, the Angus bull whose widespread use - putting him on top and bottom of the pedigrees of many Angus cattle back in the late 1990s-2000s - was a double-defect carrier, but only inherited one of those defective genes. 1680 did indeed inherit the AM (curly calf defect) from ancestors, but he himself was the 'founder' of the NH (Neuropathic Hydrocephalus) defect... the mutation that caused NH occurred in him. </p><p></p><p>I have retained and used home-bred bulls in the past with decent results, and some were pretty intensely linebred back to a specific bull, but mutations can and do occur, and just because you've bred an animal's ancestors for multiple generations, doesn't mean a deleterious gene mutation can't rear its ugly head and spread widely for several generations before two carriers happen to mate and it reveals itself.</p></blockquote><p></p>
[QUOTE="Lucky_P, post: 1648573, member: 12607"] Nick, Your supposition that this bull inherited the SCN defect isn't born out by scientific examination. This bull we purchased was, as far as we can tell, the originator of the SCN defect; that is, the mutation arose in him. While his dam and sire were not available for testing when the defective gene sequence was characterized, AI sires several generations back were tested, and found to be free of the defect, as was the single half-sib cow remaining in the herd of origin. The bull was by a son of SAF Focus of ER, out of a New Design 878 daughter... well-known genetics to Angus folks, and most of the ancestors in the 4-5 generations behind him had material on file available for testing, and all were found to be free of the SCN defect. For example, GAR Precision 1680, the Angus bull whose widespread use - putting him on top and bottom of the pedigrees of many Angus cattle back in the late 1990s-2000s - was a double-defect carrier, but only inherited one of those defective genes. 1680 did indeed inherit the AM (curly calf defect) from ancestors, but he himself was the 'founder' of the NH (Neuropathic Hydrocephalus) defect... the mutation that caused NH occurred in him. I have retained and used home-bred bulls in the past with decent results, and some were pretty intensely linebred back to a specific bull, but mutations can and do occur, and just because you've bred an animal's ancestors for multiple generations, doesn't mean a deleterious gene mutation can't rear its ugly head and spread widely for several generations before two carriers happen to mate and it reveals itself. [/QUOTE]
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In-breeding Risk same grandfather & great great grandfather
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