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Artificial Insemination (AI) for Cattle
In-breeding Risk same grandfather & great great grandfather
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<blockquote data-quote="Lucky_P" data-source="post: 1648465" data-attributes="member: 12607"><p>"By keeping my own bulls, I don't have to worry about things like curly calf syndrome, or fawn calf syndrome, or the next syndrome waiting to be announced, I know it's not in my herd."</p><p></p><p>No, that's not necessarily true. Genetic mutations occur in all herds; just because you raised a bull known not to have the defects we currently know about, doesn't mean a new one can't arise in your own herd. </p><p></p><p>Some years back, we bought a yearling reg. Angus bull, free from all known (at the time) defects. Used him for two years, then started doing AI. He was a well-behaved bull, with decent calving ease, daughters were really nice, steers were OK, so we kept and used him as a cleanup bull behind AI. for the next 6 years. </p><p>Little did we know, but he was the 'founder' for the genetic recessive Sodium Channel Neuropathy defect... a mutation that occurred in him, which he passed on to half of his offspring. </p><p>So... when he eventually ended up breeding an occasional granddaughter or daughter that didn't stick to AI... an occasional defective calf would result. They were few and far between, early on, but after the first 3 or 4, we became suspicious that there was a recessive defect at play. </p><p>We shipped him when it became evident that that he was the source... but this was before AAA and Dr. Beever (then at UofIL) had been able to characterize the genetic defect and identify the defective gene. We bled every cow/heifer in the herd, and only those that went back to that Angus bull carried the defective SCN gene; I've forgotten what the 'carrier' rate was over the entire herd, but it was pretty high.</p><p>We'd kept a nice calving-ease SimAngus son, out of a top daughter of the Angus bull to use on heifers, and as a post-AI cleanup bull. Used him one season. Had defective calves out of daughters, granddaughters, and great-granddaughters of the Angus bull - the young SimAngus bull had inherited the SCN defect from his dam, who'd also been a carrier - but we didn't know that at the time we'd selected him for use. </p><p></p><p>Since ours was a commercial herd producing mostly crossbred stock, SCN carriers sold off the farm (and their offspring) probably will never have the 'opportunity' to be crossed back to another SCN carrier, so for all intents and purposes, the SCN defect is relegated to the dustbin of history.</p></blockquote><p></p>
[QUOTE="Lucky_P, post: 1648465, member: 12607"] "By keeping my own bulls, I don’t have to worry about things like curly calf syndrome, or fawn calf syndrome, or the next syndrome waiting to be announced, I know it’s not in my herd." No, that's not necessarily true. Genetic mutations occur in all herds; just because you raised a bull known not to have the defects we currently know about, doesn't mean a new one can't arise in your own herd. Some years back, we bought a yearling reg. Angus bull, free from all known (at the time) defects. Used him for two years, then started doing AI. He was a well-behaved bull, with decent calving ease, daughters were really nice, steers were OK, so we kept and used him as a cleanup bull behind AI. for the next 6 years. Little did we know, but he was the 'founder' for the genetic recessive Sodium Channel Neuropathy defect... a mutation that occurred in him, which he passed on to half of his offspring. So... when he eventually ended up breeding an occasional granddaughter or daughter that didn't stick to AI... an occasional defective calf would result. They were few and far between, early on, but after the first 3 or 4, we became suspicious that there was a recessive defect at play. We shipped him when it became evident that that he was the source... but this was before AAA and Dr. Beever (then at UofIL) had been able to characterize the genetic defect and identify the defective gene. We bled every cow/heifer in the herd, and only those that went back to that Angus bull carried the defective SCN gene; I've forgotten what the 'carrier' rate was over the entire herd, but it was pretty high. We'd kept a nice calving-ease SimAngus son, out of a top daughter of the Angus bull to use on heifers, and as a post-AI cleanup bull. Used him one season. Had defective calves out of daughters, granddaughters, and great-granddaughters of the Angus bull - the young SimAngus bull had inherited the SCN defect from his dam, who'd also been a carrier - but we didn't know that at the time we'd selected him for use. Since ours was a commercial herd producing mostly crossbred stock, SCN carriers sold off the farm (and their offspring) probably will never have the 'opportunity' to be crossed back to another SCN carrier, so for all intents and purposes, the SCN defect is relegated to the dustbin of history. [/QUOTE]
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