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NCBA, R-CALF, COOL, USDA (No Politics!)
CDC: Tainted ground beef may be linked to 2 deaths
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<blockquote data-quote="flounder" data-source="post: 721198" data-attributes="member: 3519"><p>>>>Does cooking the meat properly kill the E coli?</p><p></p><p>>>>If yes, wouldn't it then be the fault of whoever cooked the meat?</p><p></p><p>>>>Blessings</p><p></p><p>>>>Valerie Clavin</p><p></p><p></p><p></p><p></p><p></p><p>yes it does, it kills _most_ dangerous pathogens, please see ;</p><p></p><p></p><p>How the Consumer Can Fight Foodborne Illness</p><p></p><p>The Centers for Disease Control and Prevention (CDC) recommendations for prevention of an infection caused by Escherichia coli O157:H7 include:</p><p></p><p>Cook all ground beef or hamburger thoroughly. Make sure that the cooked meat is gray or brown throughout (not pink), any juices run clear, and the inside is hot.</p><p>The temperature of the meat should reach a minimum of 160 degrees F, as measured with a digital instant-read meat thermometer.</p><p>If you are served an undercooked hamburger in a restaurant, send it back.</p><p></p><p></p><p></p><p><a href="http://www.fda.gov/AnimalVeterinary/NewsEvents/FDAVeterinarianNewsletter/ucm083980.htm" target="_blank">http://www.fda.gov/AnimalVeterinary/New ... 083980.htm</a></p><p></p><p></p><p></p><p>However, cooking does NOT kill the PrP i.e. the TSE or Transmissible Spongiform Encephalopathy aka mad cow type diseases, and there are many, with atypical TSE cases spreading. There are many here in the USA and all of North America. THE c-BSE, atypical h-BSE and l-BSE have all been documented in North America. The USDA have absolutely no idea how bad the mad cow type disease is here in the USA, and we been trading products from Canada live and dead that could very well be tainted with TSE, like two lovers swapping spit. but in my opinion, the argument of defense to the consumer by comparison, when you buy a car, you get some sort of warranty, unless it is a clunker program i.e. 'buy as is'. is that how you want your industry to be construed as i.e. clunker program? at least with a car, you can trace the owners, you can trace parts, but not with a product we are to consume each day and hope it does not kill us, and if it does, oh well, you cannot trace it ??? which brings me to the infamous USDA dead stock downer cow school lunch program, the top 'clunker program' of all ;</p><p></p><p></p><p>New studies on the heat resistance of hamster-adapted scrapie agent: Threshold survival after ashing at 600°C suggests an inorganic template of replication </p><p></p><p>Paul Brown*,dagger , Edward H. RauDagger , Bruce K. Johnson*, Alfred E. Bacote*, Clarence J. Gibbs Jr.*, and D. Carleton Gajdusek§ </p><p> </p><p>* Laboratory of Central Nervous System Studies, National Institute of Neurological Disorders and Stroke, and Dagger Environmental Protection Branch, Division of Safety, Office of Research Services, National Institutes of Health, Bethesda, MD 20892; and § Institut Alfred Fessard, Centre National de la Recherche Scientifique, 91198 Gif sur Yvette, France </p><p> </p><p>Contributed by D. Carleton Gajdusek, December 22, 1999 </p><p> </p><p>Abstract </p><p> </p><p>One-gram samples from a pool of crude brain tissue from hamsters infected with the 263K strain of hamster-adapted scrapie agent were placed in covered quartz-glass crucibles and exposed for either 5 or 15 min to dry heat at temperatures ranging from 150°C to 1,000°C. Residual infectivity in the treated samples was assayed by the intracerebral inoculation of dilution series into healthy weanling hamsters, which were observed for 10 months; disease transmissions were verified by Western blot testing for proteinase-resistant protein in brains from clinically positive hamsters. Unheated control tissue contained 9.9 log10LD50/g tissue; after exposure to 150°C, titers equaled or exceeded 6 log10LD50/g, and after exposure to 300°C, titers equaled or exceeded 4 log10LD50/g. Exposure to 600°C completely ashed the brain samples, which, when reconstituted with saline to their original weights, transmitted disease to 5 of 35 inoculated hamsters. No transmissions occurred after exposure to 1,000°C. These results suggest that an inorganic molecular template with a decomposition point near 600°C is capable of nucleating the biological replication of the scrapie agent. </p><p> </p><p>transmissible spongiform encephalopathy | scrapie | prion | medical waste | incineration </p><p> </p><p>Introduction </p><p> </p><p>The infectious agents responsible for transmissible spongiform encephalopathy (TSE) are notoriously resistant to most physical and chemical methods used for inactivating pathogens, including heat. It has long been recognized, for example, that boiling is ineffective and that higher temperatures are most efficient when combined with steam under pressure (i.e., autoclaving). As a means of decontamination, dry heat is used only at the extremely high temperatures achieved during incineration, usually in excess of 600°C. It has been assumed, without proof, that incineration totally inactivates the agents of TSE, whether of human or animal origin. It also has been assumed that the replication of these agents is a strictly biological process (1), although the notion of a "virus" nucleant of an inorganic molecular cast of the infectious beta -pleated peptide also has been advanced (2). In this paper, we address these issues by means of dry heat inactivation studies. </p><p> </p><p>see full text: </p><p> </p><p><a href="http://www.pnas.org/cgi/content/full/97/7/3418" target="_blank">http://www.pnas.org/cgi/content/full/97/7/3418</a> </p><p></p><p></p><p>PLoS ONE. 2008; 3(8): e2969. Published online 2008 August 13. doi: 10.1371/journal.pone.0002969. PMCID: PMC2493038 </p><p></p><p>Copyright This is an open-access article distributed under the terms of the Creative Commons Public Domain declaration which stipulates that, once placed in the public domain, this work may be freely reproduced, distributed, transmitted, modified, built upon, or otherwise used by anyone for any lawful purpose. </p><p></p><p>Prion Infected Meat-and-Bone Meal Is Still Infectious after Biodiesel Production </p><p></p><p>Cathrin E. Bruederle,1* Robert M. Hnasko,1 Thomas Kraemer,2 Rafael A. Garcia,3 Michael J. Haas,3 William N. Marmer,3 and John Mark Carter1 1USDA-ARS WRRC, Foodborne Contaminants Research Unit, Albany, California, United States of America 2Forensic Toxicology, Institute of Legal Medicine, Saarland University, Homburg/Saar, Germany </p><p></p><p>3USDA-ARS ERRC, Fats, Oils and Animal Coproducts Research Unit, Wyndmoor, Pennsylvania, United States of America Neil Mabbott, Editor </p><p></p><p>University of Edinburgh, United Kingdom * E-mail: mhtml:%7B33B38F65-8D2E-434D-8F9B-8BDCD77D3066%7Dmid://00000089/!x-usc:mailto:cat </p><p></p><p>Conceived and designed the experiments: CEB RMH WNM JMC. Performed the experiments: CEB RMH TK. Analyzed the data: CEB TK JMC. Contributed reagents/materials/analysis tools: CEB RMH TK RAG MJH JMC. Wrote the paper: CEB. Received April 21, 2008; Accepted July 24, 2008. </p><p></p><p><a href="http://www.pubmedcentral.nih.gov/articlerender.fcgi?artid=2493038" target="_blank">http://www.pubmedcentral.nih.gov/articl ... id=2493038</a></p><p></p><p>P04.08 Environmental Persistence of TSEs - Extraction of PrPSc from Soil Smith, A; Fernie, Karen; Somerville, R Neuropathogenesis Unit, UK Background: There are concerns about the potential spread of transmissible spongiform encephalopathies (TSEs) by environmental routes following, for example, burial of infected carcasses or the disposal of waste water. The extent to which TSE infectivity survives or is disseminated within soil and soil water is unclear as is the likelihood of ensuing infection. Aim: As part of this environmental project, soil samples are being collected from lysimeters containing either infected bovine heads or boluses of infectivity. The aim of this experiment is to devise a method for the extraction of PrPSc from soil and examines the interaction between soil and its components and TSE infectivity. Methods: Samples from two soil types (clay and sandy loam) were spiked with known amounts of TSE infected brain homogenate and subjected to various extraction methods including combinations of freeze/thaw, boiling, sonication and mixing with various solvents and detergents. Any recovery was determined on western blot using PrPSc as a surrogate marker for the presence of TSE infectivity. Results: These experiments have shown that PrPSc binds strongly to both sandy and clay soil, and to pure sand (quartz). Elution from quartz and the soils was only achieved in the presence of the detergent sarkosyl, and in the case of clay soil, satisfactory elution was only achieved if PrPSc was digested with proteinase K. This finding suggests that components in clay soil may bind differently to PrP than those of sandy soil, and that the N-terminal domain of PrP is involved in this binding. Conclusion: These results form the basis of a method for the extraction of PrPSc from soil and will be used to assay samples from a large scale lysimeter experiment. Samples testing positive for the presence of PrPSc will be selected for bioassay in mice. Results to date suggest that TSE infectivity may bind strongly to soil components and could therefore persist in the environment for long periods of time. </p><p></p><p>P04.61 </p><p></p><p>Survival of PrPSc during Simulated Wastewater Treatment Processes </p><p></p><p>Pedersen, J1; Hinckley, G1; McMahon, K2; McKenzie, D3; Aiken, JM3 1University of Wisconsin, Soil Science/Civil and Environmental Engineering, USA; 2University of Wisconsin, Civil and Environmental Engineering, USA; 3University of Wisconsin, Comparative Biosciences, USA </p><p></p><p>Concern has been expressed that prions could enter wastewater treatment systems through sewer and/or septic systems (e.g., necropsy laboratories, rural meat processors, private game dressing) or through leachate from landfills that have received TSE-contaminated material. Prions are highly resistant to degradation and many disinfection procedures raising concern that they could survive conventional wastewater treatment. Here, we report the results of experiments examining the partitioning and survival of PrPSc during simulated wastewater treatment processes including activated and mesophilic anaerobic sludge digestion. We establish that PrPSc can be efficiently extracted from activated and anaerobic digester sludges with 1% sodium dodecyl sulfate, 10% sodium undecyl sulfate, and 1% sodium N-lauryl sarcosinate. Activated sludge digestion does not result in significant degradation of PrPSc. The protein partitions strongly to the activated sludge solids and is expected to enter biosolids treatment processes. A large fraction of PrPSc survived simulated mesophilic anaerobic sludge digestion. Our results suggest that if prions were to enter municipal waste water treatment systems, most of the agent would partition to activated sludge solids, survive mesophilic anaerobic digestion, and be present in treated biosolids. Land application of biosolids containing prions could represent a route for their unintentional introduction into the environment. Our results argue for excluding inputs of prions to municipal wastewater treatment facilities that would result in unacceptable risk of prion disease transmission via contaminated biosolids. </p><p></p><p>P04.71 Oral Transmission of Prion Disease Is Enhanced by Binding to Soil Particles Johnson, C; Pedersen, J; Chappell, R; McKenzie, D; Aiken, J University of Wisconsin - Madison, USA A long-unanswered question in prion biology is how certain transmissible spongiform encephalopathies (TSEs), such as sheep scrapie and cervid chronic wasting disease, spread from animal to animal. Anecdotal evidence and controlled field experiments have suggested the presence of an environmental TSE reservoir. We, and others, have speculated that soil may harbor TSE agent in the environment and allow its transfer to naïve hosts. TSE infectivity can persist in soil for years, and we previously demonstrated that the disease-associated form of the prion protein binds to soil particles and that prions adsorbed to the common soil mineral montmorillonite (Mte) retain infectivity following intracerebral inoculation. We assessed the oral infectivity of Mte- and soil-bound prions and found that prions bound to Mte are orally bioavailable and that, unexpectedly, binding to Mte significantly enhances disease penetrance and reduces incubation period relative to unbound agent. Cox proportional hazards modelling revealed that across the doses of TSE agent tested, Mte increased the effective infectious titer by a factor of 680 relative to unbound agent. Oral exposure to Mte-associated prions led to TSE development in experimental animals even at doses too low to produce clinical symptoms in the absence of the mineral. We tested the oral infectivity of prions bound to three whole soils differing in texture, mineralogy and organic carbon content, and found soil-bound prions to be orally infectious. Two of the three soils increased oral transmission of disease, and the infectivity of agent bound to the third soil was equivalent to that of unbound agent. Enhanced infectivity of soilbound prions may explain the environmental transmission of some TSEs despite the presumably low levels shed into the environment. </p><p></p><p>P04.104 Survival of Prion Proteins in Environmental Matrices Maluquer de Motes, C1; Torres, JM2; Pumarola, M3; Girones, R1 1University of Barcelona, Spain; 2Centro de Investigacion en Sanidad Animal, Spain; 3Autonomous University of Barcelona, Spain Several publications have suggested the environment as a possible route of transmission, especially for sheep scrapie and cervid Chronic Wasting Disease (CWD). The role of the environment as a reservoir for these disorders is difficult to prove and faces a considerable lack of information. In this work, different methodologies have been developed to evaluate the survival and inactivation of TSE agents in environmental matrices. Different slaughterhouse and urban sewage samples were spiked with diverse strains of either scrapie or BSE agents and kept under controlled conditions for extended periods of time. Aliquots of every experiment were sequentially collected and concentrated according to a methodology specifically selected for each type of matrix. Sensitivity of the methods developed was estimated among 2-10 ƒÊg of infected tissue. PrPres was finally detected by western blot. Films were then transformed into digital pictures, signal intensities were quantified and regression models were computed. According to the results obtained, scrapie agent showed higher stability than BSE in all the environments studied. However, no significant differences were observed among mouse-passaged scrapie strains and sheep scrapie. The regression models provided t90 and t99 values (times of incubation necessaries for 90% and 99% reduction of PrPres levels). In urban sewage, i.e., t99 was estimated as about 50 and 22 days for scrapie and BSE respectively. In general, the effect of the matrix was clearly observed in all the experiments, showing up to a 6-8 fold higher reduction of PrPres levels in comparison to PBS controls. As some of the inocula were titrated in terms of infectious doses, we approximated the decay of PrPres levels to the reduction of infectivity for both agents. In slaughterhouse wastewater, i.e., two-log reduction was observed for both agents after 30-35 days of incubation. Data on infectivity will be confirmed by a series of bioassay experiments. </p><p></p><p>P04.125 Environmental Persistence of TSE Infectivity: Field Studies Fernie, K; Smith, A; Somerville, R Neuropathogenesis Unit, Roslin Institute, UK Background: There is concern about the consequences of contamination of the environment with TSE infectivity. Infectivity may enter the environment by various routes, persist in the ground and spread from the original source to contaminate an extended area and groundwater. Aims: We are studying this problem by addressing the following questions: 1. Does infectivity with some containment (e.g. in a carcass) survive in the carcass over time; 2. Does infectivity without containment survive, and is it disseminated into the surrounding soil and water? 3. Do the environmental conditions, e.g. soil type and pH, affect the survival and/or transport of infectivity through soil? Methods: To address these questions, we are performing two field experiments (with appropriate containment) each using two soil types. Air temperature, rainfall, soil temperature and moisture content are being monitored. In one experiment a series of 10 bovine heads have been spiked with the BSE derived TSE strain 301V and buried in the two soils, contained within individual lysimeters, for exhumation and analysis at yearly intervals. Rainwater flowing through and collected as groundwater is also being analysed. In the second experiment a bolus of infected brain is buried at the centre of two 3 meter diameter lysimeters and soil samples taken from them at regular intervals. Water flow-through is also analysed. Results: To date, the first two bovine heads have been exhumed and the surrounding soil sampled. Both of the exhumed heads were apparently largely decomposed but on examination of the brain cavity were found to contain significant amounts of brain tissue. These have been sampled and are presently being analysed. The soil samples taken from around the heads and five sets of core samples taken from the soil surrounding the buried brain in the two large lysimeters are presently being analysed for PrPSc, the abnormal protein associated with the TSEs and for infectivity. Water samples have also been collected for analyses. Discussion: We will use the acquired data to build a predictive model of TSE behaviour in the environment which will inform future risk assessments. </p><p></p><p>snip...end....NEUROPRION 2007...TSS </p><p></p><p>Public release date: 11-Aug-2008 </p><p></p><p>Contact: Dr. Björn Seidel mhtml:%7B33B38F65-8D2E-434D-8F9B-8BDCD77D3066%7Dmid://00000089/!x-usc:mailto:bjo 49-297-230-2330 Fraunhofer-Gesellschaft </p><p></p><p>Resistant prions </p><p></p><p>A flock of sheep at pasture – a seemingly idyllic scene. But appearances can be deceptive: If the animals are suffering from scrapie, entire flocks may perish. Scrapie is an infectious disease in which prions destroy the animal's brain, rather like BSE. The brain becomes porous, the sheep lose their orientation, they suffer from strong itching sensations and scrape off their fleece. Eventually, the infected animals die. </p><p></p><p>It is difficult to contain the disease – all too often, scrapie will break out again on the same farm several months or years after it has apparently been eradicated. Are the prions transmitted not only by direct contact, but also by the environment – perhaps by the pastures? How long do prions that get into the pasture via the saliva and excrements of the sick animals, persist in the ground? </p><p></p><p>Together with fellow-scientists from the Robert Koch Institute in Berlin and the Friedrich Loeffler Institute (Federal Research Institute for Animal Health) on the island of Riems, research scientists from the Fraunhofer Institute for Molecular Biology and Applied Ecology IME in Schmallenberg investigated these questions on behalf of the German Ministry for Environment, Nature Conservation and Nuclear Safety BMU. "We mixed soil samples with scrapie pathogens to find out how long the pathogens would survive," says Dr. Björn Seidel, who headed the investigations at IME. "Even after 29 months, in other words more than two years, we were still able to detect prions in the soil." But are these prions still infectious? "The soil actually seems to increase the infectiousness of the pathogens. The incubation period – the time it takes for the disease to break out – is exceedingly short even after the prions have persisted in the soil for 29 months. All of the animals that were given contaminated soil became sick within a very short time. </p><p></p><p>These results indicate that fresh incidences of scrapie among sheep are due to contaminated pastures," says Seidel in summary. The results of the study reveal that sheep may even become infected from the surface water, though the risk of infection is much lower in this case. There is no danger to humans, however: scrapie pathogens seem unable to affect them. </p><p></p><p>Another cause for concern is chronic wasting disease (CWD). Like BSE and scrapie, this is caused by prions, but it mainly affects deer. The numbers of infected animals in North America are rising steeply. How long do BSE and CWD prions survive in the ground? "To find this out, we urgently need to carry out further tests. The appropriate research applications have already been submitted," says Seidel. </p><p></p><p><a href="http://www.eurekalert.org/pub_releases/2008-08/f-rp081108.php" target="_blank">http://www.eurekalert.org/pub_releases/ ... 081108.php</a> # </p><p></p><p>snip... full text ; </p><p></p><p><a href="http://bse-atypical.blogspot.com/2008/11/resistance-of-bovine-spongiform.html" target="_blank">http://bse-atypical.blogspot.com/2008/1 ... iform.html</a></p><p></p><p></p><p></p><p></p><p>Over the next 8-10 weeks, approximately 40% of all the adult mink on the farm died from TME.</p><p></p><p>snip...</p><p></p><p>The rancher was a ''dead stock'' feeder using mostly (>95%) downer or dead dairy cattle...</p><p></p><p><a href="http://web.archive.org/web/20030516051623/http://www.bseinquiry.gov.uk/files/mb/m09/tab05.pdf" target="_blank">http://web.archive.org/web/200305160516 ... /tab05.pdf</a></p><p></p><p>Food Authorities Affected by Hallmark/Westland Meat Packing Co. Beef Recall February 2006 - February 2008</p><p></p><p><a href="http://www.fns.usda.gov/fns/safety/Hallmark-Westland_byState.pdf" target="_blank">http://www.fns.usda.gov/fns/safety/Hall ... yState.pdf</a></p><p></p><p>Members of The HSUS are also concerned about the meat products provided to their children through the National School Lunch Program. More than 31 million school children receive lunches through the program each school day. To assist states in providing healthful, low-cost or free meals, USDA provides states with various commodities including ground beef. As evidenced by the HallmarkNVestland investigation and recall, the potential for downed animals to make their way into the National School Lunch Program is neither speculative nor hypothetical.</p><p></p><p><a href="http://biotech.law.lsu.edu/cases/FDA/hsus-v-schafer-usda-complaint.pdf" target="_blank">http://biotech.law.lsu.edu/cases/FDA/hs ... plaint.pdf</a></p><p></p><p>>>>In the papers, the government alleges the meatpacking plant slaughtered and processed downer cows for nearly four years — from January 2004 to September 2007 — at the average rate of one every six weeks...<<<</p><p> </p><p><a href="http://downercattle.blogspot.com/2009/09/suit-meatpacker-used-downer-cows-for-4.html" target="_blank">http://downercattle.blogspot.com/2009/0 ... for-4.html</a></p><p></p><p>"The alleged misrepresentations by Hallmark and Westland could have impacted the health of many of our nation's most vulnerable citizens--our schoolchildren," said Tony West, Assistant Attorney General of the Justice Department's Civil Division. "Our intervention in this case demonstrates how seriously we will pursue allegations such as these."</p><p></p><p><a href="http://downercattle.blogspot.com/2009/05/us-government-sues-westlandhallmark.html" target="_blank">http://downercattle.blogspot.com/2009/0 ... lmark.html</a></p><p></p><p></p><p>Tuesday, November 17, 2009 </p><p></p><p>SEAC EFFECT OF AGE ON THE PATHOGENESIS OF TRANSMISSIBLE SPONGIFORM ENCEPHALOPATHIES SEAC 103/2</p><p></p><p></p><p><a href="http://downercattle.blogspot.com/2009/11/seac-effect-of-age-on-pathogenesis-of.html" target="_blank">http://downercattle.blogspot.com/2009/1 ... is-of.html</a></p><p></p><p></p><p></p><p>Friday, September 4, 2009</p><p></p><p>FOIA REQUEST ON FEED RECALL PRODUCT 429,128 lbs. feed for ruminant animals may have been contaminated with prohibited material Recall # V-258-2009</p><p></p><p><a href="http://madcowfeed.blogspot.com/2009/09/foia-request-on-feed-recall-product.html" target="_blank">http://madcowfeed.blogspot.com/2009/09/ ... oduct.html</a></p><p></p><p>Saturday, August 29, 2009</p><p></p><p>FOIA REQUEST FEED RECALL 2009 Product may have contained prohibited materials Bulk Whole Barley, Recall # V-256-2009</p><p></p><p><a href="http://madcowfeed.blogspot.com/2009/08/foia-request-feed-recall-2009-product.html" target="_blank">http://madcowfeed.blogspot.com/2009/08/ ... oduct.html</a></p><p></p><p>----- Original Message ----- From: "Terry S. Singeltary Sr." <flounder9@VERIZON.NET> To: <BSE-L@LISTS.AEGEE.ORG> Sent: Thursday, November 05, 2009 9:25 PM Subject: [BSE-L] re-FOIA REQUEST ON FEED RECALL PRODUCT contaminated with prohibited material Recall # V-258-2009 and Recall # V-256-2009</p><p></p><p><a href="http://madcowfeed.blogspot.com/2009/11/re-foia-request-on-feed-recall-product.html" target="_blank">http://madcowfeed.blogspot.com/2009/11/ ... oduct.html</a></p><p></p><p></p><p></p><p>Tuesday, December 1, 2009 </p><p></p><p></p><p>IMPORTATION OF CANADIAN CATTLE, BISON, SHEEP, AND GOATS INTO THE UNITED STATES 12/1/09</p><p></p><p></p><p><a href="http://usdameatexport.blogspot.com/2009/12/importation-of-canadian-cattle-bison.html" target="_blank">http://usdameatexport.blogspot.com/2009 ... bison.html</a></p><p></p><p></p><p>Monday, November 23, 2009 </p><p></p><p>BSE GBR RISK ASSESSMENTS UPDATE NOVEMBER 23, 2009 COMMISSION OF THE EUROPEAN COMMUNITIES AND O.I.E.</p><p></p><p></p><p><a href="http://docket-aphis-2006-0041.blogspot.com/2009/11/bse-gbr-risk-assessments-update.html" target="_blank">http://docket-aphis-2006-0041.blogspot. ... pdate.html</a></p><p></p><p></p><p>Wednesday, November 18, 2009 </p><p></p><p>R-CALF: 40 Groups Disagree With USDA's Latest BSE Court Submission</p><p></p><p></p><p><a href="http://bse-atypical.blogspot.com/2009/11/r-calf-40-groups-disagree-with-usdas.html" target="_blank">http://bse-atypical.blogspot.com/2009/1 ... usdas.html</a></p><p></p><p></p><p></p><p>2009 UPDATE ON ALABAMA AND TEXAS MAD COWS 2005 and 2006</p><p></p><p></p><p></p><p><a href="http://bse-atypical.blogspot.com/2006/08/bse-atypical-texas-and-alabama-update.html" target="_blank">http://bse-atypical.blogspot.com/2006/0 ... pdate.html</a></p><p></p><p></p><p></p><p></p><p>Monday, October 19, 2009</p><p></p><p>Atypical BSE, BSE, and other human and animal TSE in North America Update October 2009</p><p></p><p></p><p></p><p><a href="http://bse-atypical.blogspot.com/2009/10/atypical-bse-bse-and-other-human-and.html" target="_blank">http://bse-atypical.blogspot.com/2009/1 ... n-and.html</a></p><p></p><p></p><p></p><p></p><p>THE USA Bovine is not exempt from the TSE prion agent. you cannot hide from them, and you can only hide them for so long. you cannot kill them, and they know no boundaries or borders or age group (in terms of CJD reportable only in the 55 and or younger age groups only, this defies common sense and science). THE UKBSEnvCJD only theory is wrong, one of which that consists of only the UK bovine, that of only one strain, the human TSE there from i.e. nvCJD, and that there from of only one strain, in only one Geographical location of the globe, when the same feeding and rendering practices mirrored that of the UK, only the UK started some 5 years before we did, and scrapie, and all of the strains there from typical and atypical, all of which are right here in the USA, and have been, where humans of old age group, and young age group are dyeing from CJD, long and short incubation, some with kuru type amyloid plaques, some with the same symptoms as the nvCJD. the systematic failure of all agencies in the august 4, 1997 partial and voluntary mad cow feed ban, and the systematic failure in the enhanced BSE surveillance of 2004, all PROVEN to be such. ONE cannot say that many have not been exposed. one can only thank God, that only a few have been documented that went clinical, to date. but who will watch the children for the next 5+ decades? i'm serious, who will watch these kids that were exposed all across our Nation for 4 years or more to the dead stock downer cow school lunch program, where the most high risk cattle for BSE/TSE i.e. dead stock downers were fed to our kids??? this is what i don't understand, these parents will get their panty hose all twisted up because a few animals were abused, and do absolutely nothing about the fact there kids were abused, and in fact, in my opinion, poisoned. this is a disease that can incubate without symptoms for over 5 decades, or not, for some it's shorter, but once clinical, it is 100% fatal. with atypical TSE spreading, one can only guess of what the infectious dose is ??? I guess, hope it's minimal and forget about it, or hope everybody else does. ..i refuse too. ...</p><p></p><p></p><p></p><p></p><p></p><p>TSS</p></blockquote><p></p>
[QUOTE="flounder, post: 721198, member: 3519"] >>>Does cooking the meat properly kill the E coli? >>>If yes, wouldn't it then be the fault of whoever cooked the meat? >>>Blessings >>>Valerie Clavin yes it does, it kills _most_ dangerous pathogens, please see ; How the Consumer Can Fight Foodborne Illness The Centers for Disease Control and Prevention (CDC) recommendations for prevention of an infection caused by Escherichia coli O157:H7 include: Cook all ground beef or hamburger thoroughly. Make sure that the cooked meat is gray or brown throughout (not pink), any juices run clear, and the inside is hot. The temperature of the meat should reach a minimum of 160 degrees F, as measured with a digital instant-read meat thermometer. If you are served an undercooked hamburger in a restaurant, send it back. [url=http://www.fda.gov/AnimalVeterinary/NewsEvents/FDAVeterinarianNewsletter/ucm083980.htm]http://www.fda.gov/AnimalVeterinary/New ... 083980.htm[/url] However, cooking does NOT kill the PrP i.e. the TSE or Transmissible Spongiform Encephalopathy aka mad cow type diseases, and there are many, with atypical TSE cases spreading. There are many here in the USA and all of North America. THE c-BSE, atypical h-BSE and l-BSE have all been documented in North America. The USDA have absolutely no idea how bad the mad cow type disease is here in the USA, and we been trading products from Canada live and dead that could very well be tainted with TSE, like two lovers swapping spit. but in my opinion, the argument of defense to the consumer by comparison, when you buy a car, you get some sort of warranty, unless it is a clunker program i.e. 'buy as is'. is that how you want your industry to be construed as i.e. clunker program? at least with a car, you can trace the owners, you can trace parts, but not with a product we are to consume each day and hope it does not kill us, and if it does, oh well, you cannot trace it ??? which brings me to the infamous USDA dead stock downer cow school lunch program, the top 'clunker program' of all ; New studies on the heat resistance of hamster-adapted scrapie agent: Threshold survival after ashing at 600°C suggests an inorganic template of replication Paul Brown*,dagger , Edward H. RauDagger , Bruce K. Johnson*, Alfred E. Bacote*, Clarence J. Gibbs Jr.*, and D. Carleton Gajdusek§ * Laboratory of Central Nervous System Studies, National Institute of Neurological Disorders and Stroke, and Dagger Environmental Protection Branch, Division of Safety, Office of Research Services, National Institutes of Health, Bethesda, MD 20892; and § Institut Alfred Fessard, Centre National de la Recherche Scientifique, 91198 Gif sur Yvette, France Contributed by D. Carleton Gajdusek, December 22, 1999 Abstract One-gram samples from a pool of crude brain tissue from hamsters infected with the 263K strain of hamster-adapted scrapie agent were placed in covered quartz-glass crucibles and exposed for either 5 or 15 min to dry heat at temperatures ranging from 150°C to 1,000°C. Residual infectivity in the treated samples was assayed by the intracerebral inoculation of dilution series into healthy weanling hamsters, which were observed for 10 months; disease transmissions were verified by Western blot testing for proteinase-resistant protein in brains from clinically positive hamsters. Unheated control tissue contained 9.9 log10LD50/g tissue; after exposure to 150°C, titers equaled or exceeded 6 log10LD50/g, and after exposure to 300°C, titers equaled or exceeded 4 log10LD50/g. Exposure to 600°C completely ashed the brain samples, which, when reconstituted with saline to their original weights, transmitted disease to 5 of 35 inoculated hamsters. No transmissions occurred after exposure to 1,000°C. These results suggest that an inorganic molecular template with a decomposition point near 600°C is capable of nucleating the biological replication of the scrapie agent. transmissible spongiform encephalopathy | scrapie | prion | medical waste | incineration Introduction The infectious agents responsible for transmissible spongiform encephalopathy (TSE) are notoriously resistant to most physical and chemical methods used for inactivating pathogens, including heat. It has long been recognized, for example, that boiling is ineffective and that higher temperatures are most efficient when combined with steam under pressure (i.e., autoclaving). As a means of decontamination, dry heat is used only at the extremely high temperatures achieved during incineration, usually in excess of 600°C. It has been assumed, without proof, that incineration totally inactivates the agents of TSE, whether of human or animal origin. It also has been assumed that the replication of these agents is a strictly biological process (1), although the notion of a "virus" nucleant of an inorganic molecular cast of the infectious beta -pleated peptide also has been advanced (2). In this paper, we address these issues by means of dry heat inactivation studies. see full text: [url=http://www.pnas.org/cgi/content/full/97/7/3418]http://www.pnas.org/cgi/content/full/97/7/3418[/url] PLoS ONE. 2008; 3(8): e2969. Published online 2008 August 13. doi: 10.1371/journal.pone.0002969. PMCID: PMC2493038 Copyright This is an open-access article distributed under the terms of the Creative Commons Public Domain declaration which stipulates that, once placed in the public domain, this work may be freely reproduced, distributed, transmitted, modified, built upon, or otherwise used by anyone for any lawful purpose. Prion Infected Meat-and-Bone Meal Is Still Infectious after Biodiesel Production Cathrin E. Bruederle,1* Robert M. Hnasko,1 Thomas Kraemer,2 Rafael A. Garcia,3 Michael J. Haas,3 William N. Marmer,3 and John Mark Carter1 1USDA-ARS WRRC, Foodborne Contaminants Research Unit, Albany, California, United States of America 2Forensic Toxicology, Institute of Legal Medicine, Saarland University, Homburg/Saar, Germany 3USDA-ARS ERRC, Fats, Oils and Animal Coproducts Research Unit, Wyndmoor, Pennsylvania, United States of America Neil Mabbott, Editor University of Edinburgh, United Kingdom * E-mail: mhtml:%7B33B38F65-8D2E-434D-8F9B-8BDCD77D3066%7Dmid://00000089/!x-usc:mailto:cat Conceived and designed the experiments: CEB RMH WNM JMC. Performed the experiments: CEB RMH TK. Analyzed the data: CEB TK JMC. Contributed reagents/materials/analysis tools: CEB RMH TK RAG MJH JMC. Wrote the paper: CEB. Received April 21, 2008; Accepted July 24, 2008. [url=http://www.pubmedcentral.nih.gov/articlerender.fcgi?artid=2493038]http://www.pubmedcentral.nih.gov/articl ... id=2493038[/url] P04.08 Environmental Persistence of TSEs - Extraction of PrPSc from Soil Smith, A; Fernie, Karen; Somerville, R Neuropathogenesis Unit, UK Background: There are concerns about the potential spread of transmissible spongiform encephalopathies (TSEs) by environmental routes following, for example, burial of infected carcasses or the disposal of waste water. The extent to which TSE infectivity survives or is disseminated within soil and soil water is unclear as is the likelihood of ensuing infection. Aim: As part of this environmental project, soil samples are being collected from lysimeters containing either infected bovine heads or boluses of infectivity. The aim of this experiment is to devise a method for the extraction of PrPSc from soil and examines the interaction between soil and its components and TSE infectivity. Methods: Samples from two soil types (clay and sandy loam) were spiked with known amounts of TSE infected brain homogenate and subjected to various extraction methods including combinations of freeze/thaw, boiling, sonication and mixing with various solvents and detergents. Any recovery was determined on western blot using PrPSc as a surrogate marker for the presence of TSE infectivity. Results: These experiments have shown that PrPSc binds strongly to both sandy and clay soil, and to pure sand (quartz). Elution from quartz and the soils was only achieved in the presence of the detergent sarkosyl, and in the case of clay soil, satisfactory elution was only achieved if PrPSc was digested with proteinase K. This finding suggests that components in clay soil may bind differently to PrP than those of sandy soil, and that the N-terminal domain of PrP is involved in this binding. Conclusion: These results form the basis of a method for the extraction of PrPSc from soil and will be used to assay samples from a large scale lysimeter experiment. Samples testing positive for the presence of PrPSc will be selected for bioassay in mice. Results to date suggest that TSE infectivity may bind strongly to soil components and could therefore persist in the environment for long periods of time. P04.61 Survival of PrPSc during Simulated Wastewater Treatment Processes Pedersen, J1; Hinckley, G1; McMahon, K2; McKenzie, D3; Aiken, JM3 1University of Wisconsin, Soil Science/Civil and Environmental Engineering, USA; 2University of Wisconsin, Civil and Environmental Engineering, USA; 3University of Wisconsin, Comparative Biosciences, USA Concern has been expressed that prions could enter wastewater treatment systems through sewer and/or septic systems (e.g., necropsy laboratories, rural meat processors, private game dressing) or through leachate from landfills that have received TSE-contaminated material. Prions are highly resistant to degradation and many disinfection procedures raising concern that they could survive conventional wastewater treatment. Here, we report the results of experiments examining the partitioning and survival of PrPSc during simulated wastewater treatment processes including activated and mesophilic anaerobic sludge digestion. We establish that PrPSc can be efficiently extracted from activated and anaerobic digester sludges with 1% sodium dodecyl sulfate, 10% sodium undecyl sulfate, and 1% sodium N-lauryl sarcosinate. Activated sludge digestion does not result in significant degradation of PrPSc. The protein partitions strongly to the activated sludge solids and is expected to enter biosolids treatment processes. A large fraction of PrPSc survived simulated mesophilic anaerobic sludge digestion. Our results suggest that if prions were to enter municipal waste water treatment systems, most of the agent would partition to activated sludge solids, survive mesophilic anaerobic digestion, and be present in treated biosolids. Land application of biosolids containing prions could represent a route for their unintentional introduction into the environment. Our results argue for excluding inputs of prions to municipal wastewater treatment facilities that would result in unacceptable risk of prion disease transmission via contaminated biosolids. P04.71 Oral Transmission of Prion Disease Is Enhanced by Binding to Soil Particles Johnson, C; Pedersen, J; Chappell, R; McKenzie, D; Aiken, J University of Wisconsin - Madison, USA A long-unanswered question in prion biology is how certain transmissible spongiform encephalopathies (TSEs), such as sheep scrapie and cervid chronic wasting disease, spread from animal to animal. Anecdotal evidence and controlled field experiments have suggested the presence of an environmental TSE reservoir. We, and others, have speculated that soil may harbor TSE agent in the environment and allow its transfer to naïve hosts. TSE infectivity can persist in soil for years, and we previously demonstrated that the disease-associated form of the prion protein binds to soil particles and that prions adsorbed to the common soil mineral montmorillonite (Mte) retain infectivity following intracerebral inoculation. We assessed the oral infectivity of Mte- and soil-bound prions and found that prions bound to Mte are orally bioavailable and that, unexpectedly, binding to Mte significantly enhances disease penetrance and reduces incubation period relative to unbound agent. Cox proportional hazards modelling revealed that across the doses of TSE agent tested, Mte increased the effective infectious titer by a factor of 680 relative to unbound agent. Oral exposure to Mte-associated prions led to TSE development in experimental animals even at doses too low to produce clinical symptoms in the absence of the mineral. We tested the oral infectivity of prions bound to three whole soils differing in texture, mineralogy and organic carbon content, and found soil-bound prions to be orally infectious. Two of the three soils increased oral transmission of disease, and the infectivity of agent bound to the third soil was equivalent to that of unbound agent. Enhanced infectivity of soilbound prions may explain the environmental transmission of some TSEs despite the presumably low levels shed into the environment. P04.104 Survival of Prion Proteins in Environmental Matrices Maluquer de Motes, C1; Torres, JM2; Pumarola, M3; Girones, R1 1University of Barcelona, Spain; 2Centro de Investigacion en Sanidad Animal, Spain; 3Autonomous University of Barcelona, Spain Several publications have suggested the environment as a possible route of transmission, especially for sheep scrapie and cervid Chronic Wasting Disease (CWD). The role of the environment as a reservoir for these disorders is difficult to prove and faces a considerable lack of information. In this work, different methodologies have been developed to evaluate the survival and inactivation of TSE agents in environmental matrices. Different slaughterhouse and urban sewage samples were spiked with diverse strains of either scrapie or BSE agents and kept under controlled conditions for extended periods of time. Aliquots of every experiment were sequentially collected and concentrated according to a methodology specifically selected for each type of matrix. Sensitivity of the methods developed was estimated among 2-10 ƒÊg of infected tissue. PrPres was finally detected by western blot. Films were then transformed into digital pictures, signal intensities were quantified and regression models were computed. According to the results obtained, scrapie agent showed higher stability than BSE in all the environments studied. However, no significant differences were observed among mouse-passaged scrapie strains and sheep scrapie. The regression models provided t90 and t99 values (times of incubation necessaries for 90% and 99% reduction of PrPres levels). In urban sewage, i.e., t99 was estimated as about 50 and 22 days for scrapie and BSE respectively. In general, the effect of the matrix was clearly observed in all the experiments, showing up to a 6-8 fold higher reduction of PrPres levels in comparison to PBS controls. As some of the inocula were titrated in terms of infectious doses, we approximated the decay of PrPres levels to the reduction of infectivity for both agents. In slaughterhouse wastewater, i.e., two-log reduction was observed for both agents after 30-35 days of incubation. Data on infectivity will be confirmed by a series of bioassay experiments. P04.125 Environmental Persistence of TSE Infectivity: Field Studies Fernie, K; Smith, A; Somerville, R Neuropathogenesis Unit, Roslin Institute, UK Background: There is concern about the consequences of contamination of the environment with TSE infectivity. Infectivity may enter the environment by various routes, persist in the ground and spread from the original source to contaminate an extended area and groundwater. Aims: We are studying this problem by addressing the following questions: 1. Does infectivity with some containment (e.g. in a carcass) survive in the carcass over time; 2. Does infectivity without containment survive, and is it disseminated into the surrounding soil and water? 3. Do the environmental conditions, e.g. soil type and pH, affect the survival and/or transport of infectivity through soil? Methods: To address these questions, we are performing two field experiments (with appropriate containment) each using two soil types. Air temperature, rainfall, soil temperature and moisture content are being monitored. In one experiment a series of 10 bovine heads have been spiked with the BSE derived TSE strain 301V and buried in the two soils, contained within individual lysimeters, for exhumation and analysis at yearly intervals. Rainwater flowing through and collected as groundwater is also being analysed. In the second experiment a bolus of infected brain is buried at the centre of two 3 meter diameter lysimeters and soil samples taken from them at regular intervals. Water flow-through is also analysed. Results: To date, the first two bovine heads have been exhumed and the surrounding soil sampled. Both of the exhumed heads were apparently largely decomposed but on examination of the brain cavity were found to contain significant amounts of brain tissue. These have been sampled and are presently being analysed. The soil samples taken from around the heads and five sets of core samples taken from the soil surrounding the buried brain in the two large lysimeters are presently being analysed for PrPSc, the abnormal protein associated with the TSEs and for infectivity. Water samples have also been collected for analyses. Discussion: We will use the acquired data to build a predictive model of TSE behaviour in the environment which will inform future risk assessments. snip...end....NEUROPRION 2007...TSS Public release date: 11-Aug-2008 Contact: Dr. Björn Seidel mhtml:%7B33B38F65-8D2E-434D-8F9B-8BDCD77D3066%7Dmid://00000089/!x-usc:mailto:bjo 49-297-230-2330 Fraunhofer-Gesellschaft Resistant prions A flock of sheep at pasture – a seemingly idyllic scene. But appearances can be deceptive: If the animals are suffering from scrapie, entire flocks may perish. Scrapie is an infectious disease in which prions destroy the animal's brain, rather like BSE. The brain becomes porous, the sheep lose their orientation, they suffer from strong itching sensations and scrape off their fleece. Eventually, the infected animals die. It is difficult to contain the disease – all too often, scrapie will break out again on the same farm several months or years after it has apparently been eradicated. Are the prions transmitted not only by direct contact, but also by the environment – perhaps by the pastures? How long do prions that get into the pasture via the saliva and excrements of the sick animals, persist in the ground? Together with fellow-scientists from the Robert Koch Institute in Berlin and the Friedrich Loeffler Institute (Federal Research Institute for Animal Health) on the island of Riems, research scientists from the Fraunhofer Institute for Molecular Biology and Applied Ecology IME in Schmallenberg investigated these questions on behalf of the German Ministry for Environment, Nature Conservation and Nuclear Safety BMU. "We mixed soil samples with scrapie pathogens to find out how long the pathogens would survive," says Dr. Björn Seidel, who headed the investigations at IME. "Even after 29 months, in other words more than two years, we were still able to detect prions in the soil." But are these prions still infectious? "The soil actually seems to increase the infectiousness of the pathogens. The incubation period – the time it takes for the disease to break out – is exceedingly short even after the prions have persisted in the soil for 29 months. All of the animals that were given contaminated soil became sick within a very short time. These results indicate that fresh incidences of scrapie among sheep are due to contaminated pastures," says Seidel in summary. The results of the study reveal that sheep may even become infected from the surface water, though the risk of infection is much lower in this case. There is no danger to humans, however: scrapie pathogens seem unable to affect them. Another cause for concern is chronic wasting disease (CWD). Like BSE and scrapie, this is caused by prions, but it mainly affects deer. The numbers of infected animals in North America are rising steeply. How long do BSE and CWD prions survive in the ground? "To find this out, we urgently need to carry out further tests. The appropriate research applications have already been submitted," says Seidel. [url=http://www.eurekalert.org/pub_releases/2008-08/f-rp081108.php]http://www.eurekalert.org/pub_releases/ ... 081108.php[/url] # snip... full text ; [url=http://bse-atypical.blogspot.com/2008/11/resistance-of-bovine-spongiform.html]http://bse-atypical.blogspot.com/2008/1 ... iform.html[/url] Over the next 8-10 weeks, approximately 40% of all the adult mink on the farm died from TME. snip... The rancher was a ''dead stock'' feeder using mostly (>95%) downer or dead dairy cattle... [url=http://web.archive.org/web/20030516051623/http://www.bseinquiry.gov.uk/files/mb/m09/tab05.pdf]http://web.archive.org/web/200305160516 ... /tab05.pdf[/url] Food Authorities Affected by Hallmark/Westland Meat Packing Co. Beef Recall February 2006 - February 2008 [url=http://www.fns.usda.gov/fns/safety/Hallmark-Westland_byState.pdf]http://www.fns.usda.gov/fns/safety/Hall ... yState.pdf[/url] Members of The HSUS are also concerned about the meat products provided to their children through the National School Lunch Program. More than 31 million school children receive lunches through the program each school day. To assist states in providing healthful, low-cost or free meals, USDA provides states with various commodities including ground beef. As evidenced by the HallmarkNVestland investigation and recall, the potential for downed animals to make their way into the National School Lunch Program is neither speculative nor hypothetical. [url=http://biotech.law.lsu.edu/cases/FDA/hsus-v-schafer-usda-complaint.pdf]http://biotech.law.lsu.edu/cases/FDA/hs ... plaint.pdf[/url] >>>In the papers, the government alleges the meatpacking plant slaughtered and processed downer cows for nearly four years — from January 2004 to September 2007 — at the average rate of one every six weeks...<<< [url=http://downercattle.blogspot.com/2009/09/suit-meatpacker-used-downer-cows-for-4.html]http://downercattle.blogspot.com/2009/0 ... for-4.html[/url] "The alleged misrepresentations by Hallmark and Westland could have impacted the health of many of our nation’s most vulnerable citizens--our schoolchildren," said Tony West, Assistant Attorney General of the Justice Department’s Civil Division. "Our intervention in this case demonstrates how seriously we will pursue allegations such as these." [url=http://downercattle.blogspot.com/2009/05/us-government-sues-westlandhallmark.html]http://downercattle.blogspot.com/2009/0 ... lmark.html[/url] Tuesday, November 17, 2009 SEAC EFFECT OF AGE ON THE PATHOGENESIS OF TRANSMISSIBLE SPONGIFORM ENCEPHALOPATHIES SEAC 103/2 [url=http://downercattle.blogspot.com/2009/11/seac-effect-of-age-on-pathogenesis-of.html]http://downercattle.blogspot.com/2009/1 ... is-of.html[/url] Friday, September 4, 2009 FOIA REQUEST ON FEED RECALL PRODUCT 429,128 lbs. feed for ruminant animals may have been contaminated with prohibited material Recall # V-258-2009 [url=http://madcowfeed.blogspot.com/2009/09/foia-request-on-feed-recall-product.html]http://madcowfeed.blogspot.com/2009/09/ ... oduct.html[/url] Saturday, August 29, 2009 FOIA REQUEST FEED RECALL 2009 Product may have contained prohibited materials Bulk Whole Barley, Recall # V-256-2009 [url=http://madcowfeed.blogspot.com/2009/08/foia-request-feed-recall-2009-product.html]http://madcowfeed.blogspot.com/2009/08/ ... oduct.html[/url] ----- Original Message ----- From: "Terry S. Singeltary Sr." <flounder9@VERIZON.NET> To: <BSE-L@LISTS.AEGEE.ORG> Sent: Thursday, November 05, 2009 9:25 PM Subject: [BSE-L] re-FOIA REQUEST ON FEED RECALL PRODUCT contaminated with prohibited material Recall # V-258-2009 and Recall # V-256-2009 [url=http://madcowfeed.blogspot.com/2009/11/re-foia-request-on-feed-recall-product.html]http://madcowfeed.blogspot.com/2009/11/ ... oduct.html[/url] Tuesday, December 1, 2009 IMPORTATION OF CANADIAN CATTLE, BISON, SHEEP, AND GOATS INTO THE UNITED STATES 12/1/09 [url=http://usdameatexport.blogspot.com/2009/12/importation-of-canadian-cattle-bison.html]http://usdameatexport.blogspot.com/2009 ... bison.html[/url] Monday, November 23, 2009 BSE GBR RISK ASSESSMENTS UPDATE NOVEMBER 23, 2009 COMMISSION OF THE EUROPEAN COMMUNITIES AND O.I.E. [url=http://docket-aphis-2006-0041.blogspot.com/2009/11/bse-gbr-risk-assessments-update.html]http://docket-aphis-2006-0041.blogspot. ... pdate.html[/url] Wednesday, November 18, 2009 R-CALF: 40 Groups Disagree With USDA's Latest BSE Court Submission [url=http://bse-atypical.blogspot.com/2009/11/r-calf-40-groups-disagree-with-usdas.html]http://bse-atypical.blogspot.com/2009/1 ... usdas.html[/url] 2009 UPDATE ON ALABAMA AND TEXAS MAD COWS 2005 and 2006 [url=http://bse-atypical.blogspot.com/2006/08/bse-atypical-texas-and-alabama-update.html]http://bse-atypical.blogspot.com/2006/0 ... pdate.html[/url] Monday, October 19, 2009 Atypical BSE, BSE, and other human and animal TSE in North America Update October 2009 [url=http://bse-atypical.blogspot.com/2009/10/atypical-bse-bse-and-other-human-and.html]http://bse-atypical.blogspot.com/2009/1 ... n-and.html[/url] THE USA Bovine is not exempt from the TSE prion agent. you cannot hide from them, and you can only hide them for so long. you cannot kill them, and they know no boundaries or borders or age group (in terms of CJD reportable only in the 55 and or younger age groups only, this defies common sense and science). THE UKBSEnvCJD only theory is wrong, one of which that consists of only the UK bovine, that of only one strain, the human TSE there from i.e. nvCJD, and that there from of only one strain, in only one Geographical location of the globe, when the same feeding and rendering practices mirrored that of the UK, only the UK started some 5 years before we did, and scrapie, and all of the strains there from typical and atypical, all of which are right here in the USA, and have been, where humans of old age group, and young age group are dyeing from CJD, long and short incubation, some with kuru type amyloid plaques, some with the same symptoms as the nvCJD. the systematic failure of all agencies in the august 4, 1997 partial and voluntary mad cow feed ban, and the systematic failure in the enhanced BSE surveillance of 2004, all PROVEN to be such. ONE cannot say that many have not been exposed. one can only thank God, that only a few have been documented that went clinical, to date. but who will watch the children for the next 5+ decades? i'm serious, who will watch these kids that were exposed all across our Nation for 4 years or more to the dead stock downer cow school lunch program, where the most high risk cattle for BSE/TSE i.e. dead stock downers were fed to our kids??? this is what i don't understand, these parents will get their panty hose all twisted up because a few animals were abused, and do absolutely nothing about the fact there kids were abused, and in fact, in my opinion, poisoned. this is a disease that can incubate without symptoms for over 5 decades, or not, for some it's shorter, but once clinical, it is 100% fatal. with atypical TSE spreading, one can only guess of what the infectious dose is ??? I guess, hope it's minimal and forget about it, or hope everybody else does. ..i refuse too. ... TSS [/QUOTE]
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