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NCBA, R-CALF, COOL, USDA (No Politics!)
Canada Feds back Quebec R+D for SRM removal equipment
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<blockquote data-quote="flounder" data-source="post: 889376" data-attributes="member: 3519"><p>Over the next 8-10 weeks, approximately 40% of all the adult mink on the farm died from TME. </p><p> </p><p> </p><p>snip... </p><p> </p><p>The rancher was a ''dead stock'' feeder using mostly (>95%) downer or dead dairy cattle... </p><p> </p><p> </p><p> <a href="http://web.archive.org/web/20030516051623/http://www.bseinquiry.gov.uk/files/mb/m09/tab05.pdf" target="_blank">http://web.archive.org/web/200305160516 ... /tab05.pdf</a> </p><p> </p><p> </p><p> </p><p>2010-2011 </p><p> </p><p>When L-type BSE was inoculated into ovine transgenic mice and Syrian hamster the resulting molecular fingerprint had changed, either in the first or a subsequent passage, from L-type into C-type BSE. In addition, non-human primates are specifically susceptible for atypical BSE as demonstrated by an approximately 50% shortened incubation time for L-type BSE as compared to C-type. Considering the current scientific information available, it cannot be assumed that these different BSE types pose the same human health risks as C-type BSE or that these risks are mitigated by the same protective measures. This study will contribute to a correct definition of specified risk material (SRM) in atypical BSE. The incumbent of this position will develop new and transfer existing, ultra-sensitive methods for the detection of atypical BSE in tissue of experimentally infected cattle. </p><p> </p><p> </p><p><a href="http://www.prionetcanada.ca/detail.aspx?menu=5&dt=293380&app=93&cat1=387&tp=20&lk=no&cat2" target="_blank">http://www.prionetcanada.ca/detail.aspx ... lk=no&cat2</a> </p><p> </p><p> </p><p> </p><p>2011 Monday, September 26, 2011 </p><p> </p><p>L-BSE BASE prion and atypical sporadic CJD </p><p> </p><p> </p><p><a href="http://bse-atypical.blogspot.com/2011/09/l-bse-base-prion-and-atypical-sporadic.html" target="_blank">http://bse-atypical.blogspot.com/2011/0 ... radic.html</a> </p><p></p><p></p><p></p><p> </p><p></p><p>14th ICID International Scientific Exchange Brochure -</p><p> </p><p>Final Abstract Number: ISE.114</p><p> </p><p>Session: International Scientific Exchange</p><p> </p><p>Transmissible Spongiform encephalopathy (TSE) animal and human TSE in North America update October 2009</p><p> </p><p>T. Singeltary</p><p> </p><p>Bacliff, TX, USA</p><p> </p><p>Background:</p><p> </p><p>An update on atypical BSE and other TSE in North America. Please remember, the typical U.K. c-BSE, the atypical l-BSE (BASE), and h-BSE have all been documented in North America, along with the typical scrapie's, and atypical Nor-98 Scrapie, and to date, 2 different strains of CWD, and also TME. All these TSE in different species have been rendered and fed to food producing animals for humans and animals in North America (TSE in cats and dogs ?), and that the trading of these TSEs via animals and products via the USA and Canada has been immense over the years, decades.</p><p> </p><p>Methods:</p><p> </p><p>12 years independent research of available data</p><p> </p><p>Results:</p><p> </p><p>I propose that the current diagnostic criteria for human TSEs only enhances and helps the spreading of human TSE from the continued belief of the UKBSEnvCJD only theory in 2009. With all the science to date refuting it, to continue to validate this old myth, will only spread this TSE agent through a multitude of potential routes and sources i.e. consumption, medical i.e., surgical, blood, dental, endoscopy, optical, nutritional supplements, cosmetics etc.</p><p> </p><p>Conclusion:</p><p> </p><p>I would like to submit a review of past CJD surveillance in the USA, and the urgent need to make all human TSE in the USA a reportable disease, in every state, of every age group, and to make this mandatory immediately without further delay. The ramifications of not doing so will only allow this agent to spread further in the medical, dental, surgical arena's. Restricting the reporting of CJD and or any human TSE is NOT scientific. Iatrogenic CJD knows NO age group, TSE knows no boundaries. I propose as with Aguzzi, Asante, Collinge, Caughey, Deslys, Dormont, Gibbs, Gajdusek, Ironside, Manuelidis, Marsh, et al and many more, that the world of TSE Transmissible Spongiform Encephalopathy is far from an exact science, but there is enough proven science to date that this myth should be put to rest once and for all, and that we move forward with a new classification for human and animal TSE that would properly identify the infected species, the source species, and then the route.</p><p> </p><p><a href="http://ww2.isid.org/Downloads/14th_ICID_ISE_Abstracts.pdf" target="_blank">http://ww2.isid.org/Downloads/14th_ICID ... tracts.pdf</a> </p><p> </p><p></p><p></p><p></p><p> :deadhorse: </p><p></p><p></p><p> :bang: </p><p></p><p></p><p> :wave: :welcome: :tiphat:</p></blockquote><p></p>
[QUOTE="flounder, post: 889376, member: 3519"] Over the next 8-10 weeks, approximately 40% of all the adult mink on the farm died from TME. snip... The rancher was a ''dead stock'' feeder using mostly (>95%) downer or dead dairy cattle... [url=http://web.archive.org/web/20030516051623/http://www.bseinquiry.gov.uk/files/mb/m09/tab05.pdf]http://web.archive.org/web/200305160516 ... /tab05.pdf[/url] 2010-2011 When L-type BSE was inoculated into ovine transgenic mice and Syrian hamster the resulting molecular fingerprint had changed, either in the first or a subsequent passage, from L-type into C-type BSE. In addition, non-human primates are specifically susceptible for atypical BSE as demonstrated by an approximately 50% shortened incubation time for L-type BSE as compared to C-type. Considering the current scientific information available, it cannot be assumed that these different BSE types pose the same human health risks as C-type BSE or that these risks are mitigated by the same protective measures. This study will contribute to a correct definition of specified risk material (SRM) in atypical BSE. The incumbent of this position will develop new and transfer existing, ultra-sensitive methods for the detection of atypical BSE in tissue of experimentally infected cattle. [url=http://www.prionetcanada.ca/detail.aspx?menu=5&dt=293380&app=93&cat1=387&tp=20&lk=no&cat2]http://www.prionetcanada.ca/detail.aspx ... lk=no&cat2[/url] 2011 Monday, September 26, 2011 L-BSE BASE prion and atypical sporadic CJD [url=http://bse-atypical.blogspot.com/2011/09/l-bse-base-prion-and-atypical-sporadic.html]http://bse-atypical.blogspot.com/2011/0 ... radic.html[/url] 14th ICID International Scientific Exchange Brochure - Final Abstract Number: ISE.114 Session: International Scientific Exchange Transmissible Spongiform encephalopathy (TSE) animal and human TSE in North America update October 2009 T. Singeltary Bacliff, TX, USA Background: An update on atypical BSE and other TSE in North America. Please remember, the typical U.K. c-BSE, the atypical l-BSE (BASE), and h-BSE have all been documented in North America, along with the typical scrapie's, and atypical Nor-98 Scrapie, and to date, 2 different strains of CWD, and also TME. All these TSE in different species have been rendered and fed to food producing animals for humans and animals in North America (TSE in cats and dogs ?), and that the trading of these TSEs via animals and products via the USA and Canada has been immense over the years, decades. Methods: 12 years independent research of available data Results: I propose that the current diagnostic criteria for human TSEs only enhances and helps the spreading of human TSE from the continued belief of the UKBSEnvCJD only theory in 2009. With all the science to date refuting it, to continue to validate this old myth, will only spread this TSE agent through a multitude of potential routes and sources i.e. consumption, medical i.e., surgical, blood, dental, endoscopy, optical, nutritional supplements, cosmetics etc. Conclusion: I would like to submit a review of past CJD surveillance in the USA, and the urgent need to make all human TSE in the USA a reportable disease, in every state, of every age group, and to make this mandatory immediately without further delay. The ramifications of not doing so will only allow this agent to spread further in the medical, dental, surgical arena's. Restricting the reporting of CJD and or any human TSE is NOT scientific. Iatrogenic CJD knows NO age group, TSE knows no boundaries. I propose as with Aguzzi, Asante, Collinge, Caughey, Deslys, Dormont, Gibbs, Gajdusek, Ironside, Manuelidis, Marsh, et al and many more, that the world of TSE Transmissible Spongiform Encephalopathy is far from an exact science, but there is enough proven science to date that this myth should be put to rest once and for all, and that we move forward with a new classification for human and animal TSE that would properly identify the infected species, the source species, and then the route. [url=http://ww2.isid.org/Downloads/14th_ICID_ISE_Abstracts.pdf]http://ww2.isid.org/Downloads/14th_ICID ... tracts.pdf[/url] :deadhorse: :bang: :wave: :welcome: :tiphat: [/QUOTE]
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