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Alberta BSE caused by illegal Ontario Importation
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<blockquote data-quote="flounder" data-source="post: 625530" data-attributes="member: 3519"><p>THE USA AND CANADA BOTH RECIEVED TAINTED UK MBM I.E. GREAVES, LEGALLY TOO. and it seems the USA got about 44 tons (my files), and Canada got about 83 tons. ...TSS</p><p></p><p></p><p>UK EXPORTS OF MBM TO WORLD</p><p> </p><p><a href="http://www.bseinquiry.gov.uk/files/mb/m11g/tab05.pdf" target="_blank">http://www.bseinquiry.gov.uk/files/mb/m11g/tab05.pdf</a></p><p> </p><p>OTHERS</p><p> </p><p>BEEF AND VEAL</p><p> </p><p><a href="http://www.bseinquiry.gov.uk/files/mb/m11f/tab08.pdf" target="_blank">http://www.bseinquiry.gov.uk/files/mb/m11f/tab08.pdf</a></p><p> </p><p><a href="http://www.bseinquiry.gov.uk/files/mb/m11f/tab09.pdf" target="_blank">http://www.bseinquiry.gov.uk/files/mb/m11f/tab09.pdf</a></p><p> </p><p><a href="http://www.bseinquiry.gov.uk/files/mb/m11f/tab10.pdf" target="_blank">http://www.bseinquiry.gov.uk/files/mb/m11f/tab10.pdf</a></p><p> </p><p>LIVE CATTLE</p><p> </p><p><a href="http://www.bseinquiry.gov.uk/files/mb/m11f/tab11.pdf" target="_blank">http://www.bseinquiry.gov.uk/files/mb/m11f/tab11.pdf</a></p><p> </p><p>FATS</p><p> </p><p><a href="http://www.bseinquiry.gov.uk/files/mb/m11g/tab01.pdf" target="_blank">http://www.bseinquiry.gov.uk/files/mb/m11g/tab01.pdf</a></p><p> </p><p>EMBRYOS</p><p> </p><p><a href="http://www.bseinquiry.gov.uk/files/mb/m11g/tab03.pdf" target="_blank">http://www.bseinquiry.gov.uk/files/mb/m11g/tab03.pdf</a></p><p> </p><p>GELATIN ETC</p><p> </p><p><a href="http://www.bseinquiry.gov.uk/files/mb/m11g/tab02.pdf" target="_blank">http://www.bseinquiry.gov.uk/files/mb/m11g/tab02.pdf</a></p><p> </p><p>SEMEN</p><p> </p><p><a href="http://www.bseinquiry.gov.uk/files/mb/m11g/tab04.pdf" target="_blank">http://www.bseinquiry.gov.uk/files/mb/m11g/tab04.pdf</a></p><p> </p><p>MEAT</p><p> </p><p><a href="http://www.bseinquiry.gov.uk/files/mb/m11g/tab05.pdf" target="_blank">http://www.bseinquiry.gov.uk/files/mb/m11g/tab05.pdf</a></p><p> </p><p>CANADA</p><p> </p><p><a href="http://www.efsa.eu.int/science/efsa_scientific_reports/gbr_assessments/scr_annexes/563/sr02_biohaz02_canada_report_annex_en1.pdf" target="_blank">http://www.efsa.eu.int/science/efsa_sci ... ex_en1.pdf</a></p><p> </p><p>USA</p><p> </p><p><a href="http://www.efsa.eu.int/science/efsa_scientific_reports/gbr_assessments/scr_annexes/574/sr03_biohaz02_usa_report_annex_en1.pdf" target="_blank">http://www.efsa.eu.int/science/efsa_sci ... ex_en1.pdf</a></p><p> </p><p>MEXICO</p><p> </p><p><a href="http://www.efsa.eu.int/science/efsa_scientific_reports/gbr_assessments/scr_annexes/566/sr04_biohaz02_mexico_report_annex_en1.pdf" target="_blank">http://www.efsa.eu.int/science/efsa_sci ... ex_en1.pdf</a></p><p> </p><p> </p><p> </p><p>Wednesday, April 16, 2008 MBM, greaves, meat offal, live cattle, imports from UK to USA vs Canada "Three of four possible manufacturers supplying a protein supplement likely fed to the animal could have included meat and bone meal (MBM) as an ingredient in its formulation. One of these manufacturers was able to confirm usage of meat and bone meal in supplements and confirm a source of MBM to be one common to previous BSE investigations."</p><p> </p><p>USA AND CANADA IMPORTS OF UK CATTLE BETWEEN 1981 - 1989</p><p> </p><p>USA = 496</p><p> </p><p>CANADA = 198</p><p> </p><p>*add 14 to 198 as last UK import to Canada, 14 in 1990</p><p> </p><p><a href="http://www.inspection.gc.ca/english/sci/ahra/bseris/bserise.pdf" target="_blank">http://www.inspection.gc.ca/english/sci ... serise.pdf</a></p><p> </p><p>HERE is another look at all the imports for both the USA and Canada of UK live cattle and greaves exports ;</p><p> </p><p>UK Exports of Live Cattle by Value 1986-96</p><p> </p><p>USA 697 LIVE CATTLE</p><p> </p><p>CANADA 299 LIVE CATTLE</p><p> </p><p><a href="http://www.bseinquiry.gov.uk/files/mb/m11f/tab11.pdf" target="_blank">http://www.bseinquiry.gov.uk/files/mb/m11f/tab11.pdf</a></p><p> </p><p>UK TABLE of Exports of meal of meat and meat offal; greaves 1979 - 1995</p><p> </p><p>USA 24 TONS</p><p> </p><p>CANADA 83 TONS</p><p> </p><p><a href="http://www.bseinquiry.gov.uk/files/mb/m12/tab12.pdf" target="_blank">http://www.bseinquiry.gov.uk/files/mb/m12/tab12.pdf</a></p><p> </p><p>HOWEVER, my files show 44 tons of greaves for USA. ...TSS</p><p> </p><p>Subject: Re: exports from the U.K. of it's MBM to U.S.??? From: <a href="mailto:S.J.Pearsall@esg.maff.gsi.gov.uk">S.J.Pearsall@esg.maff.gsi.gov.uk</a> Date: Tue, 8 Feb 2000 14:03:16 +0000 To: <a href="mailto:flounder@wt.net">flounder@wt.net</a> (Receipt Notification Requested) (Non Receipt Notification Requested)</p><p> </p><p>Terry</p><p> </p><p>Meat and bonemeal is not specifically classified for overseas trade purposes. The nearest equivalent is listed as flours and meals of meat or offals (including tankage), unfit for human consumption; greaves. UK exports of this to the US are listed below:</p><p> </p><p>Country Tonnes</p><p> </p><p>1980 1981 12 1982 1983 1984 10 1985 2 1986 1987 1988 1989 20 1990</p><p> </p><p>Data for exports between 1975 and 1979 are not readily available. These can be obtained (at a charge) from data retailers appointed by HM Customs and Excise: BTSL (Tel: 01372 463121) or Abacus (01245 252222). Best wishes Simon Pearsall Overseas trade statistics Stats (C&F)C</p><p> </p><p>============ END...TSS...2008============</p><p> </p><p>P04.27</p><p> </p><p>Experimental BSE Infection of Non-human Primates: Efficacy of the Oral Route</p><p> </p><p>Holznagel, E1; Yutzy, B1; Deslys, J-P2; Lasmézas, C2; Pocchiari, M3; Ingrosso, L3; Bierke, P4; Schulz-Schaeffer, W5; Motzkus, D6; Hunsmann, G6; Löwer, J1 1Paul-Ehrlich-Institut, Germany; 2Commissariat à l´Energie Atomique, France; 3Instituto Superiore di Sanità, Italy; 4Swedish Institute for Infectious Disease control, Sweden; 5Georg August University, Germany; 6German Primate Center, Germany</p><p> </p><p>Background:</p><p> </p><p>In 2001, a study was initiated in primates to assess the risk for humans to contract BSE through contaminated food. For this purpose, BSE brain was titrated in cynomolgus monkeys.</p><p> </p><p>Aims:</p><p> </p><p>The primary objective is the determination of the minimal infectious dose (MID50) for oral exposure to BSE in a simian model, and, by in doing this, to assess the risk for humans. Secondly, we aimed at examining the course of the disease to identify possible biomarkers.</p><p> </p><p>Methods:</p><p> </p><p>Groups with six monkeys each were orally dosed with lowering amounts of BSE brain: 16g, 5g, 0.5g, 0.05g, and 0.005g. In a second titration study, animals were intracerebrally (i.c.) dosed (50, 5, 0.5, 0.05, and 0.005 mg).</p><p> </p><p>Results:</p><p> </p><p>In an ongoing study, a considerable number of high-dosed macaques already developed simian vCJD upon oral or intracerebral exposure or are at the onset of the clinical phase. However, there are differences in the clinical course between orally and intracerebrally infected animals that may influence the detection of biomarkers.</p><p> </p><p>Conclusions:</p><p> </p><p>Simian vCJD can be easily triggered in cynomolgus monkeys on the oral route using less than 5 g BSE brain homogenate. The difference in the incubation period between 5 g oral and 5 mg i.c. is only 1 year (5 years versus 4 years). However, there are rapid progressors among orally dosed monkeys that develop simian vCJD as fast as intracerebrally inoculated animals.</p><p> </p><p>The work referenced was performed in partial fulfilment of the study "BSE in primates" supported by the EU (QLK1-2002-01096).</p><p> </p><p><a href="http://www.prion2007.com/pdf/Prion%20Book%20of%20Abstracts.pdf" target="_blank">http://www.prion2007.com/pdf/Prion%20Bo ... tracts.pdf</a></p><p> </p><p>look at the table and you'll see that as little as 1 mg (or 0.001 gm) caused 7% (1 of 14) of the cows to come down with BSE;</p><p> </p><p>Risk of oral infection with bovine spongiform encephalopathy agent in primates</p><p> </p><p>Corinne Ida Lasmézas, Emmanuel Comoy, Stephen Hawkins, Christian Herzog, Franck Mouthon, Timm Konold, Frédéric Auvré, Evelyne Correia, Nathalie Lescoutra-Etchegaray, Nicole Salès, Gerald Wells, Paul Brown, Jean-Philippe Deslys Summary The uncertain extent of human exposure to bovine spongiform encephalopathy (BSE)--which can lead to variant Creutzfeldt-Jakob disease (vCJD)--is compounded by incomplete knowledge about the efficiency of oral infection and the magnitude of any bovine-to-human biological barrier to transmission. We therefore investigated oral transmission of BSE to non-human primates. We gave two macaques a 5 g oral dose of brain homogenate from a BSE-infected cow. One macaque developed vCJD-like neurological disease 60 months after exposure, whereas the other remained free of disease at 76 months. On the basis of these findings and data from other studies, we made a preliminary estimate of the food exposure risk for man, which provides additional assurance that existing public health measures can prevent transmission of BSE to man.</p><p> </p><p>snip...</p><p> </p><p>BSE bovine brain inoculum</p><p> </p><p>100 g 10 g 5 g 1 g 100 mg 10 mg 1 mg 0·1 mg 0·01 mg</p><p> </p><p>Primate (oral route)* 1/2 (50%)</p><p> </p><p>Cattle (oral route)* 10/10 (100%) 7/9 (78%) 7/10 (70%) 3/15 (20%) 1/15 (7%) 1/15 (7%)</p><p> </p><p>RIII mice (ic ip route)* 17/18 (94%) 15/17 (88%) 1/14 (7%)</p><p> </p><p>PrPres biochemical detection</p><p> </p><p>The comparison is made on the basis of calibration of the bovine inoculum used in our study with primates against a bovine brain inoculum with a similar PrPres concentration that was</p><p> </p><p>inoculated into mice and cattle.8 *Data are number of animals positive/number of animals surviving at the time of clinical onset of disease in the first positive animal (%). The accuracy of</p><p> </p><p>bioassays is generally judged to be about plus or minus 1 log. ic ip=intracerebral and intraperitoneal.</p><p> </p><p>Table 1: Comparison of transmission rates in primates and cattle infected orally with similar BSE brain inocula</p><p> </p><p>Published online January 27, 2005</p><p> </p><p><a href="http://www.thelancet.com/journal/journal.isa" target="_blank">http://www.thelancet.com/journal/journal.isa</a></p><p> </p><p>It is clear that the designing scientists must</p><p> </p><p>also have shared Mr Bradley's surprise at the results because all the dose</p><p> </p><p>levels right down to 1 gram triggered infection.</p><p> </p><p><a href="http://www.bseinquiry.gov.uk/files/ws/s145d.pdf" target="_blank">http://www.bseinquiry.gov.uk/files/ws/s145d.pdf</a></p><p> </p><p>6. It also appears to me that Mr Bradley's answer (that it would take less than say 100 grams) was probably given with the benefit of hindsight; particularly if one considers that later in the same answer Mr Bradley expresses his surprise that it could take as little of 1 gram of brain to cause BSE by the oral route within the same species. This information did not become available until the "attack rate"</p><p> </p><p>experiment had been completed in 1995/96. This was a titration experiment designed to ascertain the infective dose. A range of dosages was used to ensure that the actual result was within both a lower and an upper limit within the study and the designing scientists would not have expected all the dose levels to trigger infection. The dose ranges chosen by the most informed scientists at that time ranged from 1 gram to three times one hundred grams. It is clear that the designing scientists must have also shared Mr Bradley's surprise at the results because all the dose levels right down to 1 gram triggered infection.</p><p> </p><p><a href="http://www.bseinquiry.gov.uk/files/ws/s147f.pdf" target="_blank">http://www.bseinquiry.gov.uk/files/ws/s147f.pdf</a></p><p> </p><p></p><p> </p><p><a href="http://madcowtesting.blogspot.com/2008/04/mbm-greaves-meat-offal-live-cattle.html" target="_blank">http://madcowtesting.blogspot.com/2008/ ... attle.html</a></p><p> </p><p></p><p></p><p>TSS</p></blockquote><p></p>
[QUOTE="flounder, post: 625530, member: 3519"] THE USA AND CANADA BOTH RECIEVED TAINTED UK MBM I.E. GREAVES, LEGALLY TOO. and it seems the USA got about 44 tons (my files), and Canada got about 83 tons. ...TSS UK EXPORTS OF MBM TO WORLD [url=http://www.bseinquiry.gov.uk/files/mb/m11g/tab05.pdf]http://www.bseinquiry.gov.uk/files/mb/m11g/tab05.pdf[/url] OTHERS BEEF AND VEAL [url=http://www.bseinquiry.gov.uk/files/mb/m11f/tab08.pdf]http://www.bseinquiry.gov.uk/files/mb/m11f/tab08.pdf[/url] [url=http://www.bseinquiry.gov.uk/files/mb/m11f/tab09.pdf]http://www.bseinquiry.gov.uk/files/mb/m11f/tab09.pdf[/url] [url=http://www.bseinquiry.gov.uk/files/mb/m11f/tab10.pdf]http://www.bseinquiry.gov.uk/files/mb/m11f/tab10.pdf[/url] LIVE CATTLE [url=http://www.bseinquiry.gov.uk/files/mb/m11f/tab11.pdf]http://www.bseinquiry.gov.uk/files/mb/m11f/tab11.pdf[/url] FATS [url=http://www.bseinquiry.gov.uk/files/mb/m11g/tab01.pdf]http://www.bseinquiry.gov.uk/files/mb/m11g/tab01.pdf[/url] EMBRYOS [url=http://www.bseinquiry.gov.uk/files/mb/m11g/tab03.pdf]http://www.bseinquiry.gov.uk/files/mb/m11g/tab03.pdf[/url] GELATIN ETC [url=http://www.bseinquiry.gov.uk/files/mb/m11g/tab02.pdf]http://www.bseinquiry.gov.uk/files/mb/m11g/tab02.pdf[/url] SEMEN [url=http://www.bseinquiry.gov.uk/files/mb/m11g/tab04.pdf]http://www.bseinquiry.gov.uk/files/mb/m11g/tab04.pdf[/url] MEAT [url=http://www.bseinquiry.gov.uk/files/mb/m11g/tab05.pdf]http://www.bseinquiry.gov.uk/files/mb/m11g/tab05.pdf[/url] CANADA [url=http://www.efsa.eu.int/science/efsa_scientific_reports/gbr_assessments/scr_annexes/563/sr02_biohaz02_canada_report_annex_en1.pdf]http://www.efsa.eu.int/science/efsa_sci ... ex_en1.pdf[/url] USA [url=http://www.efsa.eu.int/science/efsa_scientific_reports/gbr_assessments/scr_annexes/574/sr03_biohaz02_usa_report_annex_en1.pdf]http://www.efsa.eu.int/science/efsa_sci ... ex_en1.pdf[/url] MEXICO [url=http://www.efsa.eu.int/science/efsa_scientific_reports/gbr_assessments/scr_annexes/566/sr04_biohaz02_mexico_report_annex_en1.pdf]http://www.efsa.eu.int/science/efsa_sci ... ex_en1.pdf[/url] Wednesday, April 16, 2008 MBM, greaves, meat offal, live cattle, imports from UK to USA vs Canada "Three of four possible manufacturers supplying a protein supplement likely fed to the animal could have included meat and bone meal (MBM) as an ingredient in its formulation. One of these manufacturers was able to confirm usage of meat and bone meal in supplements and confirm a source of MBM to be one common to previous BSE investigations." USA AND CANADA IMPORTS OF UK CATTLE BETWEEN 1981 - 1989 USA = 496 CANADA = 198 *add 14 to 198 as last UK import to Canada, 14 in 1990 [url=http://www.inspection.gc.ca/english/sci/ahra/bseris/bserise.pdf]http://www.inspection.gc.ca/english/sci ... serise.pdf[/url] HERE is another look at all the imports for both the USA and Canada of UK live cattle and greaves exports ; UK Exports of Live Cattle by Value 1986-96 USA 697 LIVE CATTLE CANADA 299 LIVE CATTLE [url=http://www.bseinquiry.gov.uk/files/mb/m11f/tab11.pdf]http://www.bseinquiry.gov.uk/files/mb/m11f/tab11.pdf[/url] UK TABLE of Exports of meal of meat and meat offal; greaves 1979 - 1995 USA 24 TONS CANADA 83 TONS [url=http://www.bseinquiry.gov.uk/files/mb/m12/tab12.pdf]http://www.bseinquiry.gov.uk/files/mb/m12/tab12.pdf[/url] HOWEVER, my files show 44 tons of greaves for USA. ...TSS Subject: Re: exports from the U.K. of it's MBM to U.S.??? From: [email=S.J.Pearsall@esg.maff.gsi.gov.uk]S.J.Pearsall@esg.maff.gsi.gov.uk[/email] Date: Tue, 8 Feb 2000 14:03:16 +0000 To: [email=flounder@wt.net]flounder@wt.net[/email] (Receipt Notification Requested) (Non Receipt Notification Requested) Terry Meat and bonemeal is not specifically classified for overseas trade purposes. The nearest equivalent is listed as flours and meals of meat or offals (including tankage), unfit for human consumption; greaves. UK exports of this to the US are listed below: Country Tonnes 1980 1981 12 1982 1983 1984 10 1985 2 1986 1987 1988 1989 20 1990 Data for exports between 1975 and 1979 are not readily available. These can be obtained (at a charge) from data retailers appointed by HM Customs and Excise: BTSL (Tel: 01372 463121) or Abacus (01245 252222). Best wishes Simon Pearsall Overseas trade statistics Stats (C&F)C ============ END...TSS...2008============ P04.27 Experimental BSE Infection of Non-human Primates: Efficacy of the Oral Route Holznagel, E1; Yutzy, B1; Deslys, J-P2; Lasmézas, C2; Pocchiari, M3; Ingrosso, L3; Bierke, P4; Schulz-Schaeffer, W5; Motzkus, D6; Hunsmann, G6; Löwer, J1 1Paul-Ehrlich-Institut, Germany; 2Commissariat à l´Energie Atomique, France; 3Instituto Superiore di Sanità, Italy; 4Swedish Institute for Infectious Disease control, Sweden; 5Georg August University, Germany; 6German Primate Center, Germany Background: In 2001, a study was initiated in primates to assess the risk for humans to contract BSE through contaminated food. For this purpose, BSE brain was titrated in cynomolgus monkeys. Aims: The primary objective is the determination of the minimal infectious dose (MID50) for oral exposure to BSE in a simian model, and, by in doing this, to assess the risk for humans. Secondly, we aimed at examining the course of the disease to identify possible biomarkers. Methods: Groups with six monkeys each were orally dosed with lowering amounts of BSE brain: 16g, 5g, 0.5g, 0.05g, and 0.005g. In a second titration study, animals were intracerebrally (i.c.) dosed (50, 5, 0.5, 0.05, and 0.005 mg). Results: In an ongoing study, a considerable number of high-dosed macaques already developed simian vCJD upon oral or intracerebral exposure or are at the onset of the clinical phase. However, there are differences in the clinical course between orally and intracerebrally infected animals that may influence the detection of biomarkers. Conclusions: Simian vCJD can be easily triggered in cynomolgus monkeys on the oral route using less than 5 g BSE brain homogenate. The difference in the incubation period between 5 g oral and 5 mg i.c. is only 1 year (5 years versus 4 years). However, there are rapid progressors among orally dosed monkeys that develop simian vCJD as fast as intracerebrally inoculated animals. The work referenced was performed in partial fulfilment of the study “BSE in primates“ supported by the EU (QLK1-2002-01096). [url=http://www.prion2007.com/pdf/Prion%20Book%20of%20Abstracts.pdf]http://www.prion2007.com/pdf/Prion%20Bo ... tracts.pdf[/url] look at the table and you'll see that as little as 1 mg (or 0.001 gm) caused 7% (1 of 14) of the cows to come down with BSE; Risk of oral infection with bovine spongiform encephalopathy agent in primates Corinne Ida Lasmézas, Emmanuel Comoy, Stephen Hawkins, Christian Herzog, Franck Mouthon, Timm Konold, Frédéric Auvré, Evelyne Correia, Nathalie Lescoutra-Etchegaray, Nicole Salès, Gerald Wells, Paul Brown, Jean-Philippe Deslys Summary The uncertain extent of human exposure to bovine spongiform encephalopathy (BSE)--which can lead to variant Creutzfeldt-Jakob disease (vCJD)--is compounded by incomplete knowledge about the efficiency of oral infection and the magnitude of any bovine-to-human biological barrier to transmission. We therefore investigated oral transmission of BSE to non-human primates. We gave two macaques a 5 g oral dose of brain homogenate from a BSE-infected cow. One macaque developed vCJD-like neurological disease 60 months after exposure, whereas the other remained free of disease at 76 months. On the basis of these findings and data from other studies, we made a preliminary estimate of the food exposure risk for man, which provides additional assurance that existing public health measures can prevent transmission of BSE to man. snip... BSE bovine brain inoculum 100 g 10 g 5 g 1 g 100 mg 10 mg 1 mg 0·1 mg 0·01 mg Primate (oral route)* 1/2 (50%) Cattle (oral route)* 10/10 (100%) 7/9 (78%) 7/10 (70%) 3/15 (20%) 1/15 (7%) 1/15 (7%) RIII mice (ic ip route)* 17/18 (94%) 15/17 (88%) 1/14 (7%) PrPres biochemical detection The comparison is made on the basis of calibration of the bovine inoculum used in our study with primates against a bovine brain inoculum with a similar PrPres concentration that was inoculated into mice and cattle.8 *Data are number of animals positive/number of animals surviving at the time of clinical onset of disease in the first positive animal (%). The accuracy of bioassays is generally judged to be about plus or minus 1 log. ic ip=intracerebral and intraperitoneal. Table 1: Comparison of transmission rates in primates and cattle infected orally with similar BSE brain inocula Published online January 27, 2005 [url=http://www.thelancet.com/journal/journal.isa]http://www.thelancet.com/journal/journal.isa[/url] It is clear that the designing scientists must also have shared Mr Bradley’s surprise at the results because all the dose levels right down to 1 gram triggered infection. [url=http://www.bseinquiry.gov.uk/files/ws/s145d.pdf]http://www.bseinquiry.gov.uk/files/ws/s145d.pdf[/url] 6. It also appears to me that Mr Bradley’s answer (that it would take less than say 100 grams) was probably given with the benefit of hindsight; particularly if one considers that later in the same answer Mr Bradley expresses his surprise that it could take as little of 1 gram of brain to cause BSE by the oral route within the same species. This information did not become available until the "attack rate" experiment had been completed in 1995/96. This was a titration experiment designed to ascertain the infective dose. A range of dosages was used to ensure that the actual result was within both a lower and an upper limit within the study and the designing scientists would not have expected all the dose levels to trigger infection. The dose ranges chosen by the most informed scientists at that time ranged from 1 gram to three times one hundred grams. It is clear that the designing scientists must have also shared Mr Bradley’s surprise at the results because all the dose levels right down to 1 gram triggered infection. [url=http://www.bseinquiry.gov.uk/files/ws/s147f.pdf]http://www.bseinquiry.gov.uk/files/ws/s147f.pdf[/url] [url=http://madcowtesting.blogspot.com/2008/04/mbm-greaves-meat-offal-live-cattle.html]http://madcowtesting.blogspot.com/2008/ ... attle.html[/url] TSS [/QUOTE]
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