New York Firm Recalls Beef Carcass That Contains (BSE)

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New York Firm Recalls Beef Carcass That Contains Prohibited Materials

Recall Release CLASS II RECALL FSIS-RC-003-2010 HEALTH RISK: LOW

Congressional and Public Affairs (202) 720-9113 Atiya Khan

WASHINGTON, January 15, 2010 - Jerry Hayes Meats Inc., a Newark Valley, N.Y., establishment is recalling approximately 490 pounds of a beef carcass that may not have had the spinal column removed, which is not compliant with regulations that require the removal of spinal cord and vertebral column from cattle over 30 months of age, the U.S. Department of Agriculture's Food Safety and Inspection Service (FSIS) announced today.

Spinal cord and vertebral column are considered a specified risk material (SRM) and must be removed from cattle over 30 months of age in accordance with FSIS regulations. SRMs are tissues that are known to contain the infective agent in cattle infected with Bovine Spongiform Encephalopathy (BSE), as well as materials that are closely associated with these potentially infective tissues. Therefore, FSIS prohibits SRMs from use as human food to minimize potential human exposure to the BSE agent.

The products subject to recall include: 1- "BEEF CARCASS," which bears the establishment number "EST. 04488 M" inside the USDA mark of inspection.

The product was packed on January 7, 2010, and sold to a single customer in New York.

The problem was discovered through routine FSIS inspection activities. FSIS has received no reports of illnesses associated with consumption of this product.

FSIS routinely conducts recall effectiveness checks to verify recalling firms notify their customers (including restaurants) of the recall and that steps are taken to make certain that the product is no longer available to consumers.

Media and consumers with questions about the recall should contact the company's owner, Jerry Hayes at (607) 642-9598.


http://www.fsis.usda.gov/News_&_Events/ ... /index.asp



The most recent assessments (and reassessments) were published in June 2005 (Table I; 18), and included the categorisation of Canada, the USA, and Mexico as GBR III. Although only Canada and the USA have reported cases, the historically open system of trade in North America suggests that it is likely that BSE is present also in Mexico.

http://www.oie.int/boutique/extrait/06heim937950.pdf


Tuesday, July 14, 2009

U.S. Emergency Bovine Spongiform Encephalopathy Response Plan Summary and BSE Red Book Date: February 14, 2000 at 8:56 am PST

WHERE did we go wrong $$$

http://madcowtesting.blogspot.com/2009/ ... iform.html


Saturday, January 2, 2010

Human Prion Diseases in the United States January 1, 2010 ***FINAL***

http://prionunitusaupdate2008.blogspot. ... tates.html


my comments to PLosone here ;

http://www.plosone.org/annotation/listT ... 31e2bfa6fd




Thursday, January 14, 2010

SAMPLE COLLECTION FROM CATTLE UNDER THE BOVINE SPONGIFORM ENCEPHALOPATHY (BSE) ONGOING SURVEILLANCE PROGRAM FSIS NOTICE 05-10 1/12/10


http://bse-atypical.blogspot.com/2010/0 ... under.html




TSS


Friday, January 15, 2010

New York Firm Recalls Beef Carcass That Contains Prohibited Materials (BSE)


http://bse-atypical.blogspot.com/2010/0 ... -that.html
 
> FSIS has received no reports of illnesses associated with consumption of this product.


Here we go again. This to me is very misleading, why don't they post the rest of the science on TSE and incubation periods ? The FSIS et al know damn good and well that your not going to eat this product and fall over dead immediately or in the next few days, weeks, months from a mad cow type disease i.e. Transmissible Spongiform Encephalopathy. It takes years, decade, and or decades for a TSE to become clinical and kill. Once the TSE is clinical, you better have your things in order, your going to die. ...TSS


Science 23 November 2001: Vol. 294. no. 5547, pp. 1726 - 1728 DOI: 10.1126/science.1066838 Reports Estimation of Epidemic Size and Incubation Time Based on Age Characteristics of vCJD in the United Kingdom

Alain-Jacques Valleron,1 Pierre-Yves Boelle,1 Robert Will,2 Jean-Yves Cesbron31 Epidemiology and Information Sciences, INSERM U444, CHU Saint-Antoine, Université Pierre et Marie Curie et Assistance Publique-Hôpitaux de Paris, 27 rue Chaligny, 75012 Paris, France. 2 National Creutzfeldt-Jakob Disease Surveillance Unit, Western General Hospital, Edinburgh EH4 2XU, UK. 3 Immunité Anti-Infectieuse JE 2236, UFR de Médecine de Grenoble, Université Joseph Fourier, Domaine de la Merci, 38706 La Tronche, France. SNIP... The distribution of the vCJD incubation period that best fits the data within the framework of our model has a mean of 16.7 years, with a standard deviation of 2.6 years. The 95% upper percentile of this distribution is 21.4 years. The 95% confidence interval (CI) of the estimates of the mean and standard deviation is relatively narrow: The 95% CI for the estimate of the mean incubation period is 12.4 to 23.2 years, and the 95% CI of the standard deviation is 0.9 to 8 years (10). The decrease in susceptibility to infection in exposed subjects older than 15 years, as estimated from the parameter , was found to be very sharp: 16% per year of age (CI: 12 to 23%). This means that, under the best fitting hypothesis, an individual aged 20 years in 1981 had 55% less risk of becoming infected than a child aged 15 years (99.9% for an individual aged 70).

http://www.sciencemag.org/

http://downercattle.blogspot.com/2008/0 ... r-cow.html

Interpretation Incubation periods of infection with human prions can exceed 50 years. In human infection with BSE prions, species-barrier effects, which are characteristic of cross-species transmission, would be expected to further increase the mean and range of incubation periods, compared with recycling of prions within species. These data should inform attempts to model variant CJD epidemiology. Affiliations a. MRC Prion Unit and Department of Neurodegenerative Disease, Institute of Neurology, University College London, London WC1N 3BG, UK b. Papua New Guinea Institute of Medical Research, Goroka, EHP, Papua New Guinea c. Centre for International Health, Curtin University, Perth, AustraliaCorrespondence to: Prof John Collinge

http://www.thelancet.com/journals/lance ... 7/abstract

http://kuru-tse.blogspot.com/2009/11/no ... riant.html

http://blogs.nature.com/news/blog/2006/ ... cubat.html



THIS goes much further than the UKBSEnvCJD hamburger eating adolescents only theory. The medical, surgical, dental arena, will/have played a key role in transmission of human and animal TSE. all iatrogenic CJD is, is sporadic CJD, until route and source is proven. a human could be sub-clinical TSE from animal TSE, and pass it on to other humans via the 'Friendly Fire' 'Pass It Forward' modes of transmission. ...


The documents below were provided by Terry S. Singeltary Sr on 8 May 2000. They are optically character read (scanned into computer) and so may contain typos and unreadable parts.


http://www.mad-cow.org/00/nov00_late_news.html#fff


TIP740203/l 0424 CONFIDENTIAL

http://www.mad-cow.org/00/may00_news.html#aaa


24 Jul 00 Trade Statistics: UK to US Compiled by Terry S.Singeltary Sr of Bacliff, Texas [Opinion (webmaster): The US has focused for years on tracing, containing, and eradicating live animal imports from the UK or other countries with acknowledged BSE like Belgium, including some 499 cattle and the Vermont sheep. This strategy does not acknowledge imports of rendered bovine products from England during the BSE period nor secondary products such as surgical catgut, which is to say surgical cowgut, or dairy cattle embryos, vaccines for veterinarian and human medicines. What has become of these? Mr. Singeltary, who lost his mother to CJD of unexplained origin a few years back and went on to became a well-known TSE activist, has tracked down voluminous pertinent import data through correspondence with UK officials and searches of government web sites. Imports of such products are frequently cited by Europeans in rating BSE risks in the US and in shutting out US exports. Many people's eyes glaze over when reviewing reams of sometimes older trade statistics. There is no proof that any of the imported products was contaminated with BSE nor if so, any evidence that any BSE product lead to infection in US livestock, surgical patients, or what not. Nonetheless, the data obtained by Mr. Singeltary establish that an appalling variety and tonnage of products that were imported by the US from the UK and othr BSE-affected countries during the peak of the BSE epidemic years.]


10 January 1990

COMMERCIAL IN CONFIDENCE

NOT FOR PUBLICATION

COMMITTEE ON SAFETY OF MEDICINES WORKING PARTY ON BOVINE SPONGIFORM ENCEPHALOPATHY

http://www.mad-cow.org/00/jul00_dont_ea ... tary7.html


WHAT about iatrogenic CJD there from these animal and human typical and atypical TSE's in the USA i.e. Friendly Fire and or 'The Pass It Forward' mode of transmission, and incubation periods there of ??? did not tell the public about that either ?


SHORT REPORT Creutzfeldt-Jakob disease 38 years after diagnostic use of human growth hormone

E A Croes, G Roks, G H Jansen, P C G Nijssen, C M van Duijn

...............................................................

J Neurol Neurosurg Psychiatry 2002;72:792-793

A 47 year old man is described who developed pathology proven Creutzfeldt-Jakob disease (CJD) 38 years after receiving a low dose of human derived growth hormone (hGH) as part of a diagnostic procedure. The patient presented with a cerebellar syndrome, which is compatible with iatrogenic CJD. This is the longest incubation period described so far for iatrogenic CJD. Furthermore, this is the first report of CJD after diagnostic use of hGH. Since the patient was one of the first in the world to receive hGH, other cases of iatrogenic CJD can be expected in the coming years.Prion diseases are potentially transmissible. Human to human transmission was first reported in 1974, when a 55 year old woman was described who developed symptoms of Creutzfeldt-Jakob disease (CJD) 18 months after a corneal transplant.1 Since then, transmission has been reported after stereotactic electroencephalographic (EEG) depth recording, human growth hormone (hGH) and gonadotrophin treatment, and dura mater transplantation.2-5 More than 267 patients with iatrogenic CJD are known today and their number is growing.6 The most important iatrogenic cause of CJD is still contaminated cadaveric hGH. Exposure to contaminated hGH occurred before 1985, when recombinant growth hormone became available. In a recent study, incubation periods in 139 patients with hGH associated CJD were found to range from 5-30 years, with a median of 12 years.6 One of the factors influencing incubation time is genotype on polymorphic codon 129 of the prion protein gene.7 The incubation time is significantly shorter in people who are homozygous for either methionine or valine on this polymorphism.7 We describe the second patient with hGH related CJD in the Netherlands. The patient developed the disease 38 years after hGH injections. To our knowledge, this is the longest incubation period described for any form of iatrogenic CJD. Further-more, our patient was not treated with hGH but only received a low dose as part of a diagnostic procedure.

snip...


http://jnnp.bmj.com/content/72/6/792.abstract


J Neurol Neurosurg Psychiatry 1994;57:757-758 doi:10.1136/jnnp.57.6.757 Research Article

Transmission of Creutzfeldt-Jakob disease to a chimpanzee by electrodes contaminated during neurosurgery.

C J Gibbs, Jr, D M Asher, A Kobrine, H L Amyx, M P Sulima, D C Gajdusek + Author AffiliationsLaboratory of Central Nervous System Studies, National Institute of Neurological Disorders and Stroke, National Institutes of Health, Bethesda, MD 20892.

Abstract

Stereotactic multicontact electrodes used to probe the cerebral cortex of a middle aged woman with progressive dementia were previously implicated in the accidental transmission of Creutzfeldt-Jakob disease (CJD) to two younger patients. The diagnoses of CJD have been confirmed for all three cases. More than two years after their last use in humans, after three cleanings and repeated sterilisation in ethanol and formaldehyde vapour, the electrodes were implanted in the cortex of a chimpanzee. Eighteen months later the animal became ill with CJD. This finding serves to re-emphasise the potential danger posed by reuse of instruments contaminated with the agents of spongiform encephalopathies, even after scrupulous attempts to clean them.

http://jnnp.bmj.com/content/57/6/757.abstract


Saturday, January 2, 2010 Human Prion Diseases in the United States January 1, 2010 ***FINAL***


http://prionunitusaupdate2008.blogspot. ... tates.html


my comments to PLosone here ;


http://www.plosone.org/annotation/listT ... 31e2bfa6fd


Saturday, January 16, 2010


Evidence For CJD TSE Transmission Via Endoscopes 1-24-3 re-Singeltary to Bramble et al


http://creutzfeldt-jakob-disease.blogsp ... n-via.html


Friday, January 15, 2010 New York Firm Recalls Beef Carcass That Contains Prohibited Materials (BSE)


http://bse-atypical.blogspot.com/2010/0 ... -that.html
 
There is one major problem with testing for BSE, it has to be done on a carcass, which means testing is done to a minimum.
So cattle with BSE, that mingle throught the supply chain, basically go un detected. This is a fact. By the time testing the carcass is done, and BSE is confirmed in an animal, it has already been through a feedlot with perhaps 500+ other cattle. With Livestock traceability, at least you are able to identify the cattle, and feedlot, that the effected BSE cattle came from, and then work on a traceback of the other cattle.

There needs to be another way to test for BSE, while the cattle is alive, though it is not cost effective to do this, which I beleive can be done, but also comes with a price tag$$$

Somebody correct me if I am wrong.

http://livestock-id.blogspot.com
 
Livestock-D":10xib3vg said:
There is one major problem with testing for BSE, it has to be done on a carcass, which means testing is done to a minimum.
So cattle with BSE, that mingle throught the supply chain, basically go un detected. This is a fact. By the time testing the carcass is done, and BSE is confirmed in an animal, it has already been through a feedlot with perhaps 500+ other cattle. With Livestock traceability, at least you are able to identify the cattle, and feedlot, that the effected BSE cattle came from, and then work on a traceback of the other cattle.

There needs to be another way to test for BSE, while the cattle is alive, though it is not cost effective to do this, which I beleive can be done, but also comes with a price tag$$$

Somebody correct me if I am wrong.

http://livestock-id.blogspot.com



what traceability ??? the USDA could not trace their backside, if they had both hands right square on both cheeks. this was proven with the herd cohorts of the Texas mad cow (second one, if you include the stumbling and staggering mad cow that went to be rendered without any test at all) and the Alabama mad cow. then they stopped testing in numbers large enough to find it, due to what they knew they would find.


the price tag for a test would be about $10 to $15 dollars last i heard. a small price in my opinion for the American consumer, and the Consumers abroad. and i am sure if they were used, the price would go down. now i know what the officials will tell you, that by testing, this is not a food safety test. bullshit, it's better knowing with what you got, than not knowing, and ignoring, what you know you have. they DO have a test, it's not validated, and it will not be validated anytime soon. this is why they bought up the rights to the late Harash Narang's urine test. they did not want it validated by anyone. CWRU got it, tweaked it a bit, and shelved it. USDA REFUSES TO LET ANYONE TEST, for they know what they will find, and that is the bottom line. ITs why the USDA stopped testing to find. ...



kind regards,
terry
 

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