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A thought for a few thinkers
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<blockquote data-quote="WalnutCrest" data-source="post: 1427844" data-attributes="member: 21715"><p>Not every myoatatin mutation expresses itself the same way. Nor every myoatatin mutation is "double muscling". </p><p></p><p>The conventional problem with "double muscling" is that some myoatatin mutations begin expressing themselves in utero and can cause great difficulty in calving. The nystatin mutations that express themselves in utero are the traditional "double muscling" mutations. </p><p></p><p>There are other mutations that don't express until the calves are on the ground. These don't fit the traditional complaint as there is no change to the calving ease of the calf or its dam.</p><p></p><p>Finally, some myoatatin mutations result in more long thin muscle fibers (which results in tender beef) and others result in the same number of muscle fibers, but their diameter is increased (tougher eating).</p></blockquote><p></p>
[QUOTE="WalnutCrest, post: 1427844, member: 21715"] Not every myoatatin mutation expresses itself the same way. Nor every myoatatin mutation is "double muscling". The conventional problem with "double muscling" is that some myoatatin mutations begin expressing themselves in utero and can cause great difficulty in calving. The nystatin mutations that express themselves in utero are the traditional "double muscling" mutations. There are other mutations that don't express until the calves are on the ground. These don't fit the traditional complaint as there is no change to the calving ease of the calf or its dam. Finally, some myoatatin mutations result in more long thin muscle fibers (which results in tender beef) and others result in the same number of muscle fibers, but their diameter is increased (tougher eating). [/QUOTE]
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